Author

Molly Dalzell

University of Miami

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Alejandro V. VillarinoiD· 3 shared John J. O’SheaiD· 2 shared Lothar G. HennighauseniD· 2 shared Luis Alberto NiveloiD· 2 shared AA Armand· 1 shared Aaron B. Schultz· 1 shared Amy E. KeatingiD· 1 shared Ana Peci· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersImmunology

Alejandro V. VillarinoORCID

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3 papers

Recent·Most cited

Asymmetry and redundancy of STAT5 paralogs across CD8+ T cell differentiation states

Svetlana Ristin, Molly Dalzell, Christopher D. Armstrong, Nisa Ilsin, Antonio Miguel Fontanella, Luis Alberto Nivelo, Lothar G. Hennighausen, John J. O’Shea, Alejandro V. Villarino

The paper shows STAT5B is dominant and functionally distinct from STAT5A in CD8+ T cells, revealing redundancy and asymmetry in their roles during differentiation.

Communications Biology · Apr 4, 2026Peer-reviewedImmunology

B12 promotes gut dysbiosis and an inflammatory microenvironment that potentiates Tet2-deficient hematopoiesis.

Peter D. Lyon, Tiffany E. Leesang, John P Brabson, KTT Do, Molly Dalzell, Luis Alberto Nivelo, Minh Quang Lam, Ana Peci, Byron Fang, Victoria Strippoli, AA Armand, Praveen Kumar Singh, Sabita Roy, Felipe Beckedorff, Alejandro V. Villarino, Luisa Cimmino

This study shows that B12 supplementation promotes gut dysbiosis and inflammation, which enhances myelopoiesis in Tet2-deficient mice, suggesting a microbiome-mediated mechanism for clonal hematopoiesis.

bioRxiv · Aug 22, 2025 · PreprintCancer Biology

STAT5B leukemic mutations, altering SH2 tyrosine 665, have opposing impacts on immune gene programs

Hye‐Kyung Lee, Jichun Chen, Rachael Laura Philips, Sung‐Gwon Lee, Xingmin Feng, Zhijie Wu, Chengyu Liu, Aaron B. Schultz, Molly Dalzell, Manja Meggendorfer, Claudia Haferlach, Foster Birnbaum, Joel A Sexton, Amy E. Keating, John J. O’Shea, , Lothar G. Hennighausen

The paper investigates two STAT5B mutations in T cell leukemias, showing one causes gain-of-function and the other loss-of-function, with distinct impacts on immune cell populations and transcriptional activity.

Life Science Alliance · Apr 14, 2025Peer-reviewedGenetics