Author

Nicole Haste

Calico Life Sciences

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioinformatics· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:benchmarking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Assay:proteomics· 2 Approach:statistical model· 2 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Fiona Elizabeth McAllisteriD· 3 shared Aleksandr GauniD· 2 shared Carmela SidrauskiiD· 2 shared Jonathon J. O’BrieniD· 2 shared Yao Liang WongiD· 2 shared Adam Baker· 1 shared Amos BaruchiD· 1 shared Ana Maria Basso· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersGenetics

Fiona Elizabeth McAllisterORCID

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Author profile →

3 papers

Recent·Most cited

Platform-dependent effects of genetic variants on plasma APOL1

Qingbo S. Wang, Jinguo Huang, Leanne JG Chan, Nicole Haste, Niclas Olsson, Aleksandr Gaun, Fiona Elizabeth McAllister, Deepthi Madhireddy, Amos Baruch, Katie M. Cardone, Rachit Kumar, Marylyn DeRiggi Ritchie, Katalin Suszták, Eugene Melamud, Anastasia Baryshnikova

The paper identifies platform-dependent effects of APOL1 variants on plasma levels measured by different proteomic technologies.

iScience · Feb 7, 2026Peer-reviewedGenetics

Precise Estimation of In Vivo Protein Turnover Rates

Jonathon J. O’Brien, Vikram Narayan, Yao Liang Wong, Phillip Seitzer, Celeste M. Sandoval, Nicole Haste, Megan Smith, Ramin Rad, Aleksandr Gaun, Adam Baker, Matthew A. Kukurugya, Baby Martin-McNulty, Chunlian Zhang, Ganesh A. Kolumam, Carmela Sidrauski, , Rochelle Buffenstein

This paper introduces a new framework to estimate in vivo protein turnover rates by addressing labeling identification and amino acid enrichment challenges, revealing differences in protein turnover between mice and naked mole-rats.

bioRxiv · Nov 10, 2020 · PreprintBioinformatics

eIF2B activator prevents neurological defects caused by a chronic Integrated Stress Response

Yao Liang Wong, Lauren LeBon, Ana Maria Basso, Kathy L. Kohlhaas, Arthur L. Nikkel, Holly M. Robb, Diana L. Donnelly‐Roberts, Janani Prakash, Andrew M. Swensen, Nimrod Daniel Rubinstein, Swathi Krishnan, Fiona Elizabeth McAllister, Nicole Haste, Jonathon J. O’Brien, Margaret A. Roy, , Carmela Sidrauski

The paper shows that a novel eIF2B activator prevents neurological defects in a mouse model of Vanishing White Matter disease by normalizing the transcriptome and proteome.

eLife · Jan 9, 2019Peer-reviewedNeuroscience