Author

P. B. Ferrell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cancer & tumor microenvironment· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Approach:deep learning· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. Dunbar· 1 shared A. Goldsborough· 1 shared A. Krishnan· 1 shared A. R. Martinez Benitez· 1 shared B. F. Frey· 1 shared B. I. Reinfeld· 1 shared B. J. Reisman· 1 shared B. O. Bachmann· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

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1 shared paperCancer Biology

A. Dunbar

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Fixation Before Dissociation Using a Deep Eutectic Solvent Preserves In Vivo States and Phospho-Signaling in Single-Cell Sequencing

S. D. Fortmann, B. F. Frey, V. S. Hanumanthu, S. Liu, A. Goldsborough, K. V. Kilchrist, P. B. Ferrell, C. T. Weaver, M. B. Grant, R. S. Welner

This study introduces a deep eutectic solvent fixation method that preserves in vivo cellular states and phospho-signaling for scRNA-seq, enabling the identification of hyperactivated Th17 T cells.

bioRxiv · Feb 13, 2023 · PreprintImmunology

Modeling clonal evolution and oncogenic dependency in vivo in the context of hematopoietic transformation

R. L. Bowman, A. Dunbar, T. Mishra, W. Xiao, M. R. Waarts, I. Fernandez Maestre, S. E. Eisman, L. Cai, S. F. Cai, P. Sanchez Vela, S. Mowla, A. R. Martinez Benitez, Y. Park, I. S. Csete, A. Krishnan, , R. L. Levine

This study models clonal evolution and oncogenic dependency in hematopoietic transformation using reversible mutagenesis and allelic frameworks.

bioRxiv · May 18, 2022 · PreprintCancer Biology

A Family of Glycosylated Macrolides Selectively Target Energetic Vulnerabilities in Leukemia

B. J. Reisman, H. Guo, H. E. Ramsey, M. T. Wright, B. I. Reinfeld, P. B. Ferrell, G. A. Sulikowski, W. K. Rathmell, M. R. Savona, L. Plate, J. L. Rubinstein, B. O. Bachmann

This study identifies the mitochondrial ATP synthase as the target of apoptolidin family glycomacrolides and demonstrates their efficacy in suppressing leukemia progression.

bioRxiv · May 31, 2021Biochemistry