Author

P. Fullwood

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Cell Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 4 Approach:experimental assay· 4 Organism:human· 4 Assay:proteomics· 4 Research Focus:cancer & tumor microenvironment· 3 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1

Frequent Co-Authors

C. Francavilla· 5 shared J. Watson· 4 shared M. P. Smith· 3 shared D. Knight· 2 shared H. R. Ferguson· 2 shared J. Ferguson· 2 shared J. Haworth· 2 shared J. Parsons· 2 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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5 shared papersCancer Biology

C. Francavilla

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Proteomics Identifies Osteomodulin as a Promoter of Breast Cancer Bone Metastasis via CDK1 Activation

S. Cabral, J. Parsons, H. Harrison, T. Kedward, P. Fullwood, K. Spence, D. Lefley, D. Barden, J. Haworth, J. Watson, K. Tsafou, C. Behan, M. J. Dunning, N. Ali, B. Gyorffy, , R. Clarke

The paper identifies osteomodulin as a promoter of breast cancer bone metastasis through CDK1 activation using proteomics and phosphoproteomics analysis of patient-derived tumors.

bioRxiv · Nov 3, 2023 · PreprintCancer Biology

Oncogenic ERRB2 signals through the AP-1 transcription factor to control mesenchymal-like properties of oesophageal adenocarcinoma

S. Ogden, I. Ahmed, P. Fullwood, The OCCAMS Consortium, C. Francavilla, A. Sharrocks

This study identifies AP-1 as a key transcription factor mediating ERBB2-driven mesenchymal-like properties in oesophageal adenocarcinoma through integrated phosphoproteomics, open chromatin profiling, and transcriptome analysis.

bioRxiv · Aug 14, 2022Cancer Biology

Spatially Resolved Phosphoproteomics Reveals Fibroblast Growth Factor Receptor Recycling-driven Regulation of Autophagy and Survival

J. Watson, H. R. Ferguson, R. M. Brady, J. Ferguson, P. Fullwood, H. Mo, K. H. Bexley, D. Knight, J.-M. Schwartz, M. P. Smith, C. Francavilla

The paper uses spatially resolved phosphoproteomics to show FGFR2b recycling activates mTOR and ULK1, suppressing autophagy and promoting cell survival in cancer cells.

bioRxiv · Dec 4, 2021Cell Biology

Reciprocal Priming between RTKs at Recycling Endosomes Orchestrates Cellular Signaling Outputs

M. P. Smith, H. R. Ferguson, J. Ferguson, E. Zindy, K. M. Kowalczyk, T. Kedward, C. Bates, J. Parsons, J. Watson, S. Chandler, P. Fullwood, S. Warwood, D. Knight, R. B. Clarke, C. Francavilla

The paper identifies reciprocal priming between FGFRs and EGFR at recycling endosomes that coordinates signaling outputs in breast cancer cells.

bioRxiv · Jan 4, 2021Cell Biology

DGAT1 is a lipid metabolism oncoprotein that enables cancer cells to accumulate fatty acid while avoiding lipotoxicity

D. J. Wilcock, A. P. Badrock, R. Owen, M. Guerin, A. D. Southam, H. Johnston, S. Ogden, P. Fullwood, J. Watson, H. Ferguson, J. Haworth, D. A. Richardson, G. R. Lloyd, A. Jankevics, W. B. Dunn, , A. F. L. Hurlstone

This study identifies DGAT1 as an oncoprotein that enables cancer cells to accumulate fatty acids while avoiding lipotoxicity, promoting melanoma formation.

bioRxiv · Jun 23, 2020 · PreprintCancer Biology