Author

S. Rodriguez-Perales

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:cell type annotation· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell-cell communication· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

B. Oldrini· 2 shared M. Squatrito· 2 shared R. Torres-Ruiz· 2 shared A. Curiel-Garcia· 1 shared A. Gomez-Caturla· 1 shared A. Gomez-Moron· 1 shared A. Hernandez-Nunez· 1 shared A. Osorio· 1 shared

Activity by Year

2017-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersCancer Biology

B. Oldrini

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Phosphatases of regenerating liver balance F-actin rearrangements during T cell activation through WD repeat containing protein 1

O. Aguilar-Sopena, C. Carrasco-Padilla, A. Gomez-Moron, P. Castro-Sanchez, M. Estaras-Hermosel, S. Valvo, S. Alegre-Gomez, J. Redondo-Munoz, R. Torres, S. Rodriguez-Perales, S. Ismail, F. Sanchez-Madrid, N. B. Martin-Cofreces, M. L. Dustin, P. R. Navarro

This study shows that PRLs regulate F-actin dynamics during T cell activation by interacting with WDR1, affecting IS formation and cytokine production.

bioRxiv · Dec 28, 2023 · PreprintImmunology

Bilallelic germline mutations in MAD1L1 induce a novel syndrome of aneuploidy with high tumor susceptibility

C. Villarroya-Beltri, A. Osorio, R. Torres-Ruiz, D. Gomez-Sanchez, M. Trakala, A. Sanchez-Belmonte, F. Mercadillo, B. Pitarch, A. Hernandez-Nunez, A. Gomez-Caturla, D. Rueda, J. Perea, S. Rodriguez-Perales, M. Malumbres, M. Urioste

This study identifies biallelic MAD1L1 mutations causing aneuploidy and high tumor susceptibility, with systemic inflammation and clonal expansions observed in patient blood cells.

bioRxiv · Aug 8, 2022 · PreprintCancer Biology

MGMT genomic rearrangements contribute to chemotherapy resistance in gliomas

B. Oldrini, N. Vaquero-Siguero, Q. Mu, P. Kroon, Y. Zhang, M. Galan-Ganga, Z. Bao, Z. Wang, H. Liu, J. K. Sa, J. Zhao, H. Kim, S. Rodriguez-Perales, D.-H. Nam, R. G. Verhaak, , M. Squatrito

This study shows that MGMT genomic rearrangements cause TMZ resistance in gliomas and can be detected in exosomes as potential recurrence markers.

Nature Communications · Aug 4, 2020Peer-reviewedCancer Biology

Somatic genome editing with the RCAS/TVA-CRISPR/Cas9 system for precision tumor modeling

B. Oldrini, A. Curiel-Garcia, C. Marques, V. Matia, O. Uluckan, R. Torres-Ruiz, S. Rodriguez-Perales, J. Huse, M. Squatrito

This paper develops a mouse model using RCAS/TVA-CRISPR/Cas9 for in vivo somatic genome editing to generate accurate cancer models.

bioRxiv · Aug 14, 2017Cancer Biology