Author

V. Chevee

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Microbiology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:genome assembly· 1 Assay:metabolomics· 1 Approach:statistical model· 1

Frequent Co-Authors

A. S. Gupta· 2 shared D. S. Schneider· 2 shared N. M. Davis· 2 shared A. Louie· 1 shared A. S. Kirosingh· 1 shared A. Y. Lee· 1 shared C. L. Richards· 1 shared D. A. Portnoy· 1 shared

Activity by Year

2020-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersMicrobiology

A. S. Gupta

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Variation in the response to malaria in Diversity Outbred mice

A. S. Gupta, V. Chevee, A. S. Kirosingh, N. M. Davis, D. S. Schneider

The paper investigates genetic variation in the host response to malaria in Diversity Outbred mice by identifying loci associated with parasite load and mortality.

bioRxiv · Sep 29, 2021 · PreprintMicrobiology

Metabolomic analysis of diverse mice reveals hepatic arginase-1 as source of plasma arginase in Plasmodium chabaudi infection

N. M. Davis, M. M. Lissner, L. M. Massis, C. L. Richards, V. Chevee, A. S. Gupta, F. C. Gherardini, D. M. Monack, D. S. Schneider

The paper uses metabolomic analysis in diverse mice to show that liver damage during Plasmodium chabaudi infection releases hepatic arginase-1 into plasma, contributing to arginine depletion.

bioRxiv · Jun 11, 2021Pathology

Role of the transcriptional regulator SP140 in resistance to bacterial infections via repression of type I interferons

D. X. Ji, K. C. Witt, D. I. Kotov, S. R. Margolis, A. Louie, V. Chevee, K. J. Chen, M. Gaidt, H. S. Dhaliwal, A. Y. Lee, S. Nishimura, D. S. Zamboni, I. Kramnik, D. A. Portnoy, K. H. Darwin, R. E. Vance

This study identifies SP140 as a repressor of type I interferons that enhances resistance to bacterial infections like Legionella pneumophila and Mycobacterium tuberculosis.

bioRxiv · Jan 7, 2020Immunology