Author

Zoe Loewenthal

University Hospital Heidelberg

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Molecular Biology· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:metagenomics· 1 Research Focus:protein design· 1

Frequent Co-Authors

Mirko VölkersiD· 3 shared Norbert FreyiD· 3 shared Verena Kamuf‐Schenk· 3 shared Frank SteiniD· 2 shared Lonny JürgenseniD· 2 shared Mandy RetteliD· 2 shared Tobias JakobiiD· 2 shared Abel Martín-Garrido· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersMolecular Biology

Mirko VölkersORCID

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3 papers

Recent·Most cited

Muscle LIM Protein is a Non-Canonical RNA-Binding Protein that Engages in RNA-Dependent Cytoskeletal Assemblies

Paul Sigrist, Dimitrios Destounis, Venkatraman Ravi, Sudeep Sahadevan, Thileepan Sekaran, Jonas Bohnlein, Verena Kamuf‐Schenk, Zoe Loewenthal, Dunja Ferring–Appel, Frank Stein, Mandy Rettel, Etienne Boileau, Timon Seeger, Christoph Dieterich, Norbert Frey, , Mirko Völkers

This study identifies MLP as a non-canonical RNA-binding protein in cardiomyocytes and shows its RNA-dependent interaction with cytoskeletal complexes.

bioRxiv · Mar 15, 2026 · PreprintMolecular Biology

Ythdf2 regulates cardiac remodeling through its m6A-mRNA target transcripts

Vivien Kmietczyk, J. Oelschläger, P. Gupta, Eshita Varma, S. Hartl, Jennifer Furkel, Mathias H. Konstandin, Alexander Marx, Zoe Loewenthal, Verena Kamuf‐Schenk, Lonny Jürgensen, C. Stroh, Agnieszka A. Gorska, Abel Martín-Garrido, Joerg Heineke, , Mirko Völkers

This study shows that Ythdf2, an m6A reader protein, regulates cardiac function by controlling gene expression through its target transcripts, leading to cardiac remodeling in heart failure models.

Journal of Molecular and Cellular Cardiology · Jun 12, 2023Peer-reviewedMolecular Biology

Transient inhibition of translation improves long-term cardiac function after ischemia/reperfusion by attenuating the inflammatory response

Christoph Hofmann, Adrian Serafin, Ole M. Schwerdt, Fereshteh Sadat Younesi, Florian Sicklinger, Ingmar Sören Meyer, Ellen Malovrh, Clara Sandmann, Lonny Jürgensen, Verena Kamuf‐Schenk, Claudia Stroh, Zoe Loewenthal, Mandy Rettel, Frank Stein, Hugo Albert Katus, , Mirko Völkers

This study shows that temporarily inhibiting protein translation after heart attack reduces inflammation and improves long-term heart function by targeting the mTORC1-4EBP1-eIF4F pathway.

bioRxiv · Jul 25, 2022 · PreprintMolecular Biology