Jillian R. Haney

This study identifies genes linked to brain activity that show disrupted associations in autism spectrum disorder, highlighting excitation-inhibition imbalance and regional effects.

This study identifies transcriptomic signatures of human astrocytes across age, sex, and disease states, revealing changes in synaptic function, ionic homeostasis, and tumor microenvironment interactions.

The paper investigates the role of the complement system and C4A in schizophrenia by analyzing gene co-expression networks and finds that synaptic pathways, not the complement system, are linked to schizophrenia risk.

The paper identifies widespread transcriptomic dysregulation across multiple cortical regions in ASD, showing regional variation and genetic enrichment in synaptic and protein folding genes.

The paper identifies a convergent molecular subtype in ASD by integrating epigenomic and transcriptomic data, revealing shared dysregulation and links to immune processes and genetic risk variants.

This study shows that maintaining human pluripotent stem cells with feeders improves cortical organoid formation by activating TGFβ signaling and naive pluripotency genes, which can be mimicked with TGFβ growth factors.