Grant W. Hennig

This study shows that endothelial calcium signaling spreads widely during vascular injury, but phosphatidylserine externalization remains localized, revealing spatial uncoupling between calcium signaling and phospholipid scrambling.

This study investigates how GqPCR activity regulates detrusor smooth muscle phasic contractions and transient pressure events through effects on Ca2+ activity and membrane potential.

This study shows adenosine and low oxygen reduce bladder contractility via A2B receptors and BKCa channels in mice.

The paper investigates how extracellular Ca2+ shields endothelial cells from histone-induced injury and enables membrane repair through direct interaction with histones and membrane phospholipids.

The paper investigates the emergence of coordinated intestinal motility in human fetal development, identifying calcium dynamics and purinergic signaling as key drivers.

This study shows that pathogenic tau peptides reduce endothelial calcium signaling and vasodilation in brain microvessels, impairing cerebral blood flow.

The paper shows that P2Y1 receptors in perisynaptic Schwann cells mediate calcium signaling, which affects muscle fatigue by modulating potassium levels.