Laurie Kathleen Svoboda

This study shows that DDE exposure alters cell state and stem cell markers in breast cells, inducing an epidermal phenotype and upregulating keratinization pathways.

This study shows that lead exposure during iPSC-derived cardiomyocyte differentiation affects electrophysiological properties in a cell line- and concentration-dependent manner, with effects persisting after exposure ends.

The paper investigates sex-specific molecular responses to early-life toxicant exposures in mice across multiple life stages using transcriptomic and epigenomic data.

This study shows that PFOA exposure during cardiomyocyte differentiation alters gene expression and electrophysiological function in hiPSC-CM, with effects lasting after exposure stops.

This study shows that lead exposure during human iPSC-derived cardiomyocyte differentiation alters gene expression and pathway dysregulation, with persistent transcriptional changes even after exposure stops.

This study investigates how chemical exposures affect biological processes linked to aggressive breast cancer using transcriptomic analysis of normal breast cells from diverse donors.

This study examines how different levels of lead exposure affect neural differentiation in SH-SY5Y cells, showing significant changes in protein expression and cell morphology.

This study examines how perinatal exposure to lead or DEHP affects age-related DNA methylation and gene expression in mouse hearts, revealing sex-specific differences.

This study shows perinatal lead exposure causes sex-specific DNA methylation changes in adult mouse liver and blood, with some methylation sites overlapping between tissues.