Author

Renato Arnese

Research Institute of Molecular PathologyORCID 0000-0002-0606-8466Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:C. elegans· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Alessandra PisciottaniiD· 1 shared Alessandro ProvenzaniiD· 1 shared Alessandro QuattroneiD· 1 shared Anton MeinhartiD· 1 shared Aurora BadaloniiD· 1 shared Beatriz Escobar-DonceliD· 1 shared Cecilia PerrucciiD· 1 shared Daniele PeroniiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperMolecular Biology

Alessandra PisciottaniORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintMay 15, 2025

Disruption of ribosome dynamics and mRNA homeostasis triggers a cascading imbalance in protein synthesis in models of Amyotrophic Lateral Sclerosis

Fabio Lauria, Federica Maniscalco, Cecilia Perrucci…·Gabriella Viero

This study investigates how TDP-43 dysregulation disrupts protein synthesis in ALS models by altering ribosome dynamics and mRNA homeostasis, revealing a conserved mechanism across in vitro and in vivo systems.

DOI 10.1101/2025.05.15.654227

humanproteomics

𝕏 PostBluesky

Cell BiologybioRxivPreprintFeb 10, 2025

A fluorescent folding reporter uncovers myosin misfolding as a driver of Hypertrophic Cardiomyopathy

Renato Arnese, Philipp J. Dexheimer, Beatriz Escobar-Doncel…·Tim Clausen

This study identifies myosin misfolding as a driver of HCM using a fluorescent reporter and C. elegans model, showing dietary restrictions can reverse defects via autophagy.

DOI 10.1101/2025.02.10.636595

C. elegansexperimental assay

𝕏 PostBluesky