Author

Mitsuyoshi Nakao

Institute of Molecular Embryology and Genetics, Kumamoto UniversityORCID 0000-0002-2196-8673Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:development & morphogenesis· 1 Organism:Drosophila· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Ban SatoiD· 1 shared Daisuke SaitoiD· 1 shared Elena RyzhiiiD· 1 shared Gaoxin MaoiD· 1 shared Hideaki MorishitaiD· 1 shared Hikaru TsuchiyaiD· 1 shared Kiito Otsubo· 1 shared Kuniaki SaitoiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperDevelopmental Biology

Ban SatoORCID

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2 papers

Recent·Most cited

Repressive S-adenosylmethionine biosynthesis status inhibits transcription of HeT-A retrotransposon in the germline of Drosophila.

Yoshiki Hayashi, Shinjiro Hino, Tetsuya Sato, Soshiro Kashio, Kiito Otsubo, Kuniaki Saito, Ban Sato, Natsuko Kawano, Daisuke Saito, Masayuki Miura, Mikita Suyama, Mitsuyoshi Nakao, Satoru Kobayashi

The paper shows that repressive SAM biosynthesis in Drosophila germline inhibits HeT-A retrotransposon transcription via 6mA methylation.

The Journal of Biochemistry · Jul 2, 2025Peer-reviewedDevelopmental Biology

KEAP1 retention in phase-separated p62 bodies drives liver damage in autophagy-deficient conditions

Shuhei Takada, Nozomi Shinomiya, Gaoxin Mao, Hikaru Tsuchiya, Tomoaki Koga, Satoko Komatsu‐Hirota, Yu‐shin Sou, Manabu Abe, Elena Ryzhii, Michitaka Suzuki, Mitsuyoshi Nakao, Satoshi Waguri, Hideaki Morishita, Masaaki Komatsu

The paper shows that KEAP1 retention in p62 bodies and NRF2 activation cause liver damage when autophagy is impaired, and that blocking p62-KEAP1 interactions reduces this damage.

EMBO Reports · May 28, 2025Peer-reviewedCell Biology