Author

Dmytro Kukhtar

Institut d'Investigació Biomédica de BellvitgeORCID 0000-0002-2934-745XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 2 Developmental Biology· 1

Signature Assays, Organisms & Methods

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Organism:C. elegans· 3 Approach:experimental assay· 3 Research Focus:gene regulatory network inference· 3 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:drug response / drug discovery· 1 Approach:statistical model· 1

Frequent Co-Authors

Julián CeróniD· 3 shared David BrenaiD· 2 shared Abul Bashar Mir Md. Khademul IslamiD· 1 shared Albert García LópeziD· 1 shared Alejandro RubioiD· 1 shared Anna BigasiD· 1 shared Antonio J. GiráldeziD· 1 shared Antonio J. Pérez‐PulidoiD· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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3 shared papersGenetics

Julián CerónORCID

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3 papers

Recent·Most cited

GeneticsNature CommunicationsPeer-reviewedMay 12, 2022

Genome editing in animals with minimal PAM CRISPR-Cas9 enzymes

Jeremy Vicencio, Carlos Sánchez-Bolaños, Ismael Moreno-Sánchez…·Miguel A. Moreno-Mateos

This study demonstrates the use of SpG and SpRY CRISPR-Cas9 variants in zebrafish and C. elegans for genome editing, showing improved accessibility to previously uneditable sites.

48 citationstop 5%

zebrafishC. elegans

𝕏 PostBluesky

Developmental BiologyScientific ReportsPeer-reviewedSep 30, 2020

Ancestral function of Inhibitors-of-kappaB regulates Caenorhabditis elegans development

David Brena, Joan Bertran, Montserrat Porta-de-la-Riva…·Lluı́s Espinosa

This study identifies a role for C. elegans I{kappa}B homologs in chromatin regulation, showing they bind histones, Polycomb proteins, and alter chromatin marks, suggesting ancestral I{kappa}B functions in development.

19 citations

C. elegansstatistical model

𝕏 PostBluesky

GeneticsbioRxivPreprintAug 31, 2019

Splicing-related Retinitis Pigmentosa mutations mimicked in C. elegans allow the identification of disease modifiers and drug screens

Dmytro Kukhtar, Karinna Rubio-Peña, Xènia Serrat, Julián Cerón

The paper uses CRISPR in C. elegans to mimic human splicing factor mutations causing retinitis pigmentosa, identifies disease modifiers, and screens for drugs, finding harmful compounds.

DOI 10.1101/752956

C. elegansexperimental assay

𝕏 PostBluesky