Author

C. B. M. Porter

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:mouse· 2 Assay:scRNA-seq· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Approach:deep learning· 1 Assay:DNA methylation· 1

Frequent Co-Authors

A. Regev· 2 shared D. Dionne· 2 shared O. Ashenberg· 2 shared S. J. Riesenfeld· 2 shared A. E. Neumann· 1 shared A. G. Patel· 1 shared A. Jarret· 1 shared A. Karlstrom· 1 shared

Activity by Year

2018-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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2 shared papersImmunology

A. Regev

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Author profile →

3 papers

Recent·Most cited

A spatial cell atlas of neuroblastoma reveals developmental, epigenetic and spatial axis of tumor heterogeneity

A. G. Patel, O. Ashenberg, N. B. Collins, A. Segerstolpe, S. Jiang, M. Slyper, X. Huang, C. Caraccio, H. Jin, H. Sheppard, K. Xu, T.-C. Chang, B. A. Orr, A. Shirinifard, R. H. Chapple, , S. J. Rodig

This paper creates a spatial cell atlas of neuroblastoma, revealing tumor heterogeneity through multi-omics data and identifying spatial compartmentalization and signaling patterns.

bioRxiv · Jan 7, 2024 · PreprintCancer Biology

Parental-fetal interplay of immune genes leads to intrauterine growth restriction

G. Kaur, C. B. M. Porter, O. Ashenberg, J. Lee, S. J. Riesenfeld, M. Hofree, M. Aggelakopoulou, A. Subramanian, S. B. Kuttikkatte, K. E. Attfield, C. A. E. Desel, J. L. Davies, H. G. Evans, I. Avraham-Davidi, L. T. Nguyen, , L. Fugger

This study identifies a maternal-fetal immune interaction involving KIR2DL1 and HLA-C*05 that causes intrauterine growth restriction in a humanised mouse model.

bioRxiv · Mar 26, 2021Immunology

Skin inflammation driven by differentiation of quiescent tissue-resident ILCs into a spectrum of pathogenic effectors

P. Bielecki, S. J. Riesenfeld, M. S. Kowalczyk, M. C. Amezcua Vesely, L. Kroehling, P. Yaghoubi, D. Dionne, A. Jarret, H. R. Steach, H. M. McGee, C. B. M. Porter, P. Licona-Limon, W. Bailis, R. Jackson, N. Gagliani, , R. A. Flavell

The paper shows that psoriasis is driven by the reprogramming of quiescent skin-resident ILCs into pathogenic effectors through epigenetic and transcriptional changes.

bioRxiv · Nov 12, 2018Immunology