Author

Daqi Xu

Genentech

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Aaron Wong· 1 shared Alexander ArlanticoiD· 1 shared Alexander R. AbbasiD· 1 shared Alyaa M. Abdel‐HaleemiD· 1 shared Andréy S. ShawiD· 1 shared Brad A. FriedmaniD· 1 shared Cary D. AustiniD· 1 shared Caspar GlockiD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

Aaron Wong

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Combined antagonism of Oncostatin M (OSM) and Interleukin 6 (IL-6) provides both anti-fibrotic and anti-inflammatory benefit in pulmonary fibrosis

Rojo A. Ratsimandresy, Emma Doran, Hart S. Dengler, Sarah Emily Headland, Christopher J. Wedeles, Riccardo Guidi, Daqi Xu, Jiatong Liu, Yongchang Shi, Jianyong Wang, Salil Uttarwar, Alexander R. Abbas, Tianhe Sun, Daryle J. DePianto, Katrina B. Morshead, , Mark S. Wilson

This study shows that blocking OSM and IL-6 reduces fibrosis and inflammation in pulmonary fibrosis by targeting their shared receptor and distinct pathways.

bioRxiv · Jan 13, 2026 · PreprintImmunology

TPL2 kinase activity regulates microglial inflammatory responses and promotes neurodegeneration in tauopathy mice

Yuanyuan Wang, Tiffany Z. Wu, Ming‐Chi Tsai, Mitchell G. Rezzonico, Alyaa M. Abdel‐Haleem, Luke Xie, Vineela D. Gandham, Hai Ngu, Kimberly L. Stark, Caspar Glock, Daqi Xu, Oded Foreman, Brad A. Friedman, Morgan Sheng, Jesse E. Hanson

This study shows that TPL2 kinase activity in microglia drives neuroinflammation and neurodegeneration in tauopathy models, suggesting it as a therapeutic target.

eLife · Jul 31, 2023Peer-reviewedNeuroscience