Author

G. F. M. Watts

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 4 Cell Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 5 Approach:statistical model· 5 Assay:bulk RNA-seq· 4 Research Focus:cell type annotation· 4 Research Focus:differential expression· 3 Assay:scRNA-seq· 3 Research Focus:cell-cell communication· 2 Approach:deep learning· 2

Frequent Co-Authors

S. Raychaudhuri· 5 shared K. Wei· 4 shared F. Zhang· 3 shared M. B. Brenner· 3 shared A. H. Jonsson· 2 shared D. A. Rao· 2 shared I. Korsunsky· 2 shared J. H. Anolik· 2 shared

Activity by Year

2018-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

5 shared papersImmunology

S. Raychaudhuri

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Adipocytes regulate fibroblast function, and their loss contributes to fibroblast dysfunction in inflammatory diseases

H. Faust, T.-Y. Cheng, I. Korsunsky, G. F. M. Watts, S. T. Gal-Oz, W. Trim, K. Kongthong, A. H. Jonsson, D. P. Simmons, F. Zhang, R. Padera, S. Chubinskaya, Accelerating Medicines Partnership Program, K. Wei, S. Raychaudhuri, , M. B. Brenner

The paper shows adipocytes regulate fibroblast function through cortisol signaling, and their loss leads to fibroblast dysfunction in inflammatory diseases.

bioRxiv · May 16, 2023Cell Biology

The Chromatin Landscape of Pathogenic Transcriptional Cell States in Rheumatoid Arthritis

K. Weinand, S. Sakaue, A. Nathan, A. H. Jonsson, F. Zhang, G. F. M. Watts, Z. Zhu, Accelerating Medicines Partnership Program: RA and SLE Network, D. A. Rao, J. H. Anolik, M. B. Brenner, L. T. Donlin, K. Wei, S. Raychaudhuri

The paper identifies chromatin classes in RA synovial tissue and links them to transcriptional cell states and genetic variants.

bioRxiv · Apr 7, 2023 · PreprintImmunology

IL-1-driven stromal-neutrophil interaction in deep ulcers defines a pathotype of therapy non-responsive inflammatory bowel disease

M. Friedrich, M. Pohin, M. A. Jackson, I. Korsunsky, S. Bullers, K. Rue-Albrecht, Z. Christoforidou, D. Santhananthan, R. Ravindran, R. Sanches Peres, H. Sharpe, K. Wei, G. F. M. Watts, E. H. Mann, A. Geremia, , F. M. Powrie

bioRxiv · Feb 5, 2021 · PreprintImmunology

Defining Inflammatory Cell States in Rheumatoid Arthritis Joint Synovial Tissues by Integrating Single-cell Transcriptomics and Mass Cytometry

F. Zhang, K. Wei, K. Slowikowski, C. Y. Fonseka, D. A. Rao, S. Kelly, S. M. Goodman, D. Tabechian, L. B. Hughes, K. Salomon-Escoto, G. F. M. Watts, W. Apruzzese, D. J. Lieb, D. L. Boyle, A. M. Mandelin, , S. Raychaudhuri

The paper identifies key inflammatory cell states in RA synovial tissues using integrated single-cell and mass cytometry data from patient samples.

bioRxiv · Jun 20, 2018Immunology

A genome-wide innateness gradient defines the functional state of human innate T cells

M. Gutierrez-Arcelus, N. Teslovich, A. R. Mola, H. Kim, S. Hannes, K. Slowikowski, G. F. M. Watts, M. Brenner, S. Raychaudhuri, P. J. Brennan

The paper identifies an innateness gradient in human T cells, showing a trade-off between effector function and proliferative capacity.

bioRxiv · Mar 13, 2018Immunology