Author

J. Turk

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Assay:metabolomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

A. Garg· 1 shared A. J. Lusis· 1 shared A. Pinteric· 1 shared A. R. Lucas· 1 shared A. R. Verkerke· 1 shared A. Varsani· 1 shared A. Wilson· 1 shared B. Estifanos· 1 shared

Activity by Year

2019-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperMicrobiology

A. Garg

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Treatment with anti-inflammatory viral serpin modulates immuno-thrombotic responses and improves outcomes in SARS-CoV-2 infected mice

L. Zhang, Y. H. Li, K. Kibler, S. Kraberger, A. Varsani, J. Turk, N. Elmadbouly, E. Aliskevich, L. Spaccarelli, B. Estifanos, J. Enow, I. R. Zanetti, N. Saldevar, E. Lim, K. Browder, , A. R. Lucas

This study shows that PEGSerp-1 treatment reduces inflammation and improves outcomes in SARS-CoV-2 infected mice by modulating immuno-thrombotic responses.

bioRxiv · Sep 9, 2022 · PreprintMicrobiology

Inositol Phosphorlyceramide Synthase null Leishmania major (Δipcs-) are viable and virulent in animal infections where salvage of host sphingomyelin predominates

F. M. Kuhlmann, P. N. Key, S. M. Hickerson, J. Turk, F.-F. Hsu, S. M. Beverley

This study shows that Leishmania major lacking inositol phosphorlyceramide synthase (IPCS) remains viable and virulent in mice, suggesting SM salvage compensates for IPC loss.

bioRxiv · Jun 14, 2022Microbiology

The Lands cycle modulates plasma membrane lipid organization and insulin sensitivity in skeletal muscle

P. J. Ferrara, X. Rong, J. A. Maschek, A. R. Verkerke, P. Siripoksup, H. Song, K. C. Krishnan, J. M. Johnson, J. Turk, J. A. Houmard, A. J. Lusis, J. E. Cox, S. R. Shaikh, P. Tontonoz, K. Funai

The study identifies LPCAT3 as a key regulator of plasma membrane lipid organization and insulin sensitivity in skeletal muscle, showing its role in modulating lipid metabolism and insulin action.

bioRxiv · Dec 23, 2019 · PreprintPhysiology