Author

M. Prentki

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Assay:multiome· 1

Frequent Co-Authors

A. Al-Mass· 1 shared A. E. Jones· 1 shared A. Petcherski· 1 shared A. S. Divakaruni· 1 shared B. E. Corkey· 1 shared B. R. Desousa· 1 shared C. Mendes Ferreira· 1 shared E. J. Levens· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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1 shared paperBiochemistry

A. Al-Mass

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

The isoforms of pyruvate kinase act as nutrient sensors for the β-cell KATP channel

H. R. Foster, T. Ho, E. Potapenko, S. M. Sdao, S. L. Lewandowski, H. R. VanDeusen, S. M. Davidson, R. L. Cardone, M. Prentki, R. G. Kibbey, M. J. Merrins

This study identifies PKm1 and PKm2 as nutrient sensors that regulate β-cell KATP channels through mitochondrial PEP, influencing insulin secretion and Ca2+ oscillations.

bioRxiv · Feb 9, 2022 · PreprintCell Biology

A glycerol shunt functions as a glucose excess security valve in pancreatic β-cells

A. Al-Mass, P. Poursharifi, M.-L. Peyot, R. Lussier, E. J. Levens, J. Guida, Y. Mugabo, E. Possik, R. Ahmad, F. Al-Mulla, R. Sladek, S. R. M. Madiraju, M. Prentki

This study identifies a glycerol shunt in pancreatic β-cells that mitigates glucose excess by diverting glucose carbons to glycerol production, thereby preventing insulin hypersecretion and glucotoxicity.

bioRxiv · Nov 20, 2021 · PreprintBiochemistry

Blocking mitochondrial pyruvate import causes energy wasting via futile lipid cycling in brown fat

M. Veliova, C. Mendes Ferreira, I. Y. Benador, A. E. Jones, B. R. Desousa, K. Mahdaviani, R. Acin-Perez, A. Petcherski, A. S. Divakaruni, M. Prentki, B. E. Corkey, M. Liesa, M. F. Oliveira, O. S. Shirihai

The paper shows that blocking mitochondrial pyruvate import increases ATP synthesis via futile lipid cycling in brown adipocytes, enhancing energy expenditure without adrenergic stimulation.

bioRxiv · Nov 15, 2019 · PreprintBiochemistry