Author

N. Peng

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Biophysics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 2 Organism:bacteria (other)· 1 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Research Focus:drug response / drug discovery· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. A. Amarilla· 1 shared A. A. Khromykh· 1 shared A. Slonchak· 1 shared A. Suhrbier· 1 shared B. Yang· 1 shared C. Wu· 1 shared D. Lin· 1 shared D. Watterson· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperMicrobiology

A. A. Amarilla

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

MolPhase: An Advanced Phase Separation Predictor and an Investigation of Phytobacterial Effector in Plant

Q. Liang, N. Peng, Y. Xie, N. Kumar, W. Gao, Y. Miao

MolPhase predicts protein phase separation using physicochemical features and experimental data, showing T3Es undergo homotypic phase separation in vitro and in vivo.

bioRxiv · Sep 21, 2023 · PreprintBiophysics

Zika virus noncoding RNA cooperates with the viral protein NS5 to inhibit STAT1 phosphorylation and facilitate viral pathogenesis

A. Slonchak, X. Wang, H. Chaggar, J. Aguado, M. Freney, K. Yan, F. J. Torres, A. A. Amarilla, R. Balea, J. D. J. Sng, Y. X. Setoh, N. Peng, D. Watterson, E. Wolvetang, A. Suhrbier, A. A. Khromykh

This study identifies how ZIKV sfRNA interacts with NS5 to inhibit STAT1 phosphorylation, promoting viral pathogenesis in placental and neural tissues.

bioRxiv · May 18, 2021Microbiology

Aminoglycoside antibiotics inhibit mycobacteriophage infection

Z. Jiang, J. Wei, N. Peng, Y. Li

This study shows that certain aminoglycoside antibiotics inhibit mycobacteriophage infection while others do not, suggesting a mechanism involving the amino sugar group.

Antibiotics · Oct 19, 2020Peer-reviewedMicrobiology

Reprogramming the endogenous type III-A CRISPR-Cas system for genome editing, RNA interference and CRISPRi screening in Mycobacterium tuberculosis

K. Rahman, M. Jamal, X. Chen, W. Zhou, B. Yang, Y. Zou, W. Xu, Y. Lei, C. Wu, X. Cao, R. Tyagi, M. A. Naeem, D. Lin, Z. Habib, N. Peng, , G. Cao

This paper reprograms the endogenous CRISPR-Cas10 system in Mycobacterium tuberculosis for efficient gene editing, RNA interference, and genome-wide screening, enabling functional genomics studies and drug/vaccine development.

bioRxiv · Mar 9, 2020 · PreprintMicrobiology