Author

Petra Fallier‐Becker

University of Tübingen

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Henner KochiD· 2 shared Alexander BrackiD· 1 shared Ana GarciaiD· 1 shared Andrea SantuyiD· 1 shared Ann‐Kristin ReinholdiD· 1 shared Anna Schaedler· 1 shared Aslihan Ugun‐KlusekiD· 1 shared Benjamin Schmidt· 1 shared

Activity by Year

2019-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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2 shared papersNeuroscience

Henner KochORCID

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Author profile →

3 papers

Recent·Most cited

Axonopathy and altered synaptic development in early hippocampal epileptogenesis of Dravet syndrome

Nikolas Layer, Peter Müller, Maya Ayash, Friederike Pfeiffer, Meret Saile, Fabian Klopfer, Stefano Iavarone, Andrea Santuy, Petra Fallier‐Becker, Ulrike B. S. Hedrich, Holger Lerche, Henner Koch, Thomas V. Wuttke

This study uses single-cell RNA sequencing in a Dravet syndrome mouse model to identify early transcriptomic changes and axonopathy in interneurons during epileptogenesis.

bioRxiv · Oct 4, 2023 · PreprintNeuroscience

Claudin-12 deficiency causes nerve barrier breakdown, mechanical hypersensitivity and painful neuropathy

Jeremy Tsung-Chieh Chen, Xiawei Hu, Kathrin Doppler, Olga Breitkreuz-Korff, Isabel U. C. Otto, Joachim Schwabe, Ann‐Kristin Reinhold, Dorothee Günzel, Sophie Dithmer, Mohammed K. Hankir, Petra Fallier‐Becker, Lars Winkler, Rosel Blasig, Claudia Sommer, Alexander Brack, , Heike Lydia Rittner

This study identifies claudin-12's role in maintaining myelin and blood-nerve barriers, showing its deficiency causes mechanical hypersensitivity and painful neuropathy in male mice.

bioRxiv · Nov 26, 2019 · PreprintNeuroscience

PINK1 Regulates Dopamine and Lipids at Mitochondria to Maintain Synapses and Neuronal Function

Christine Bus, Sven Geisler, Marita Pauline Feldkaemper, Hector Flores‐Romero, Anna Schaedler, Katharina I. Zittlau, Maria Zarani, Betül Uysal, Nicolas Casadei, Petra Fallier‐Becker, Lisa Schwarz, Jos F.H.M. Brouwers, Henner Koch, Aslihan Ugun‐Klusek, Klaudia K. Maruszczak, , Julia Catherine Fitzgerald

This study identifies PINK1's role in regulating mitochondrial function, lipid metabolism, and dopamine homeostasis in dopaminergic neurons, showing that cholesterol depletion can reverse PINK1-related phenotypes.

bioRxiv · Oct 22, 2019 · PreprintNeuroscience