Author

Lisa Schwarz

Hertie Institute for Clinical Brain Research

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

Alexander RoeschiD· 1 shared Ana GarciaiD· 1 shared Anna Schaedler· 1 shared Aslihan Ugun‐KlusekiD· 1 shared Benjamin Schmidt· 1 shared Bente SiebelsiD· 1 shared Betül Uysal· 1 shared Boris Alexander HadaschikiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

Alexander RoeschORCID

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2 papers

Recent·Most cited

Lactate-dependent metabolic rewiring is associated with CD163+ macrophage polarization in human pyelonephritis

Lars Borgards, Hannah Voß, Stephanie Tautges, Bente Siebels, Christoph Krisp, Devon Siemes, Philippa Spangenberg, Jenny Bottek, Sarah Scharfenberg, J. J. F. Schmitz, Lisa Schwarz, Christian Rehme, Boris Alexander Hadaschik, Jan Hinrich Braesen, Alexander Roesch, , Daniel Robert Engel

This study identifies lactate as a metabolic driver of CD163+ macrophage polarization in human pyelonephritis, linking lactate metabolism to immune dysfunction.

bioRxiv · Apr 13, 2026 · PreprintImmunology

PINK1 Regulates Dopamine and Lipids at Mitochondria to Maintain Synapses and Neuronal Function

Christine Bus, Sven Geisler, Marita Pauline Feldkaemper, Hector Flores‐Romero, Anna Schaedler, Katharina I. Zittlau, Maria Zarani, Betül Uysal, Nicolas Casadei, Petra Fallier‐Becker, Lisa Schwarz, Jos F.H.M. Brouwers, Henner Koch, Aslihan Ugun‐Klusek, Klaudia K. Maruszczak, , Julia Catherine Fitzgerald

This study identifies PINK1's role in regulating mitochondrial function, lipid metabolism, and dopamine homeostasis in dopaminergic neurons, showing that cholesterol depletion can reverse PINK1-related phenotypes.

bioRxiv · Oct 22, 2019 · PreprintNeuroscience