Author

Radha Paturu

University of Michigan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Arul M. ChinnaiyaniD· 3 shared Rahul MannaniD· 3 shared Gabriel CruziD· 2 shared Rohit MehraiD· 2 shared Somnath Mahapatra· 2 shared Stephanie J. MineriD· 2 shared Xuhong CaoiD· 2 shared Yizhi Cao· 2 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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3 shared papersCancer Biology

Arul M. ChinnaiyanORCID

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3 papers

Recent·Most cited

A druggable chromatin vulnerability in suppressive neutrophils restores antitumor immunity

Fan Yang, Yi Bao, Yuanyuan Qiao, Gabriel Cruz, Eleanor Young, Rahul Mannan, Yuping Zhang, Matt Goodrich, Yizhi Cao, Jie Luo, Radha Paturu, Somnath Mahapatra, Jean Ching-Yi Tien, Royce Xu, Mikoto Kobayashi, , Arul M. Chinnaiyan

This paper identifies a druggable chromatin vulnerability in suppressive neutrophils that can restore antitumor immunity through mSWI/SNF antagonists.

bioRxiv · Sep 28, 2026 · PreprintCancer Biology

A Stress-Adaptive Lipid Kinase Axis Defines Metabolic Vulnerabilities in Neuroendocrine Prostate Cancer

Yang Zheng, Caleb Cheng, Yizhi Cao, Gabriel Cruz, Yuping Zhang, Radha Paturu, Somnath Mahapatra, Jing Hu, Rahul Mannan, Hüseyin Karabürk, Rupam Bhattacharyya, Yitong Yin, Yi Zhao, Wenyan Liu, Xuhong Cao, , Yuanyuan Qiao

This study identifies a stress-lipid kinase axis in NEPC driven by PIKfyve, revealing a synthetic vulnerability between PIKfyve and FASN that can be exploited for therapeutic targeting.

Cancer Cell · Aug 3, 2026Peer-reviewedCancer Biology

NNMT Loss Drives Cancer Progression by enhancing SAM availability for mTORC1 Signaling and Chromatin Methylation

Qu Deng, Erick Mitchell-Velasquez, Sharan Venkatesh, Rahul Mannan, Hyein Cho, Mohammed Alhusayan, Aqsa Yashfeen, Ramakrishnan Natesan, Natarajan V. Bhanu, Radha Paturu, Javed Siddique, Rohit Mehra, Sooryanarayana Varambally, Benjamin Garcia, David M. Feldser, , Irfan Ahmed Asangani

This study identifies NNMT as a tumor suppressor in prostate cancer that regulates SAM availability, mTORC1 signaling, and chromatin methylation, revealing a therapeutic strategy using dietary methionine restriction and mTORC1 inhibition.

bioRxiv · Jul 1, 2026 · PreprintCancer Biology