Author

S. A. Patel

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 2 Cancer Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 4 Assay:bulk RNA-seq· 3 Research Focus:cell type annotation· 3 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

J. T. Chang· 2 shared A. K. Singh· 1 shared A. R. Miller· 1 shared A. Vera Ponce de Leon· 1 shared A. Zheng· 1 shared B. Zhou· 1 shared C. E. Shields· 1 shared C. Luo· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 12 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersImmunology

J. T. Chang

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Selective IL-27 production by intestinal regulatory T cells permits gut-specific regulation of Th17 immunity

C.-H. Lin, C.-J. Wu, S. Cho, R. Patkar, L.-L. Lin, M.-C. Chen, E. Israelsson, J. Betts, M. Niedzielska, S. A. Patel, H. G. Duong, R. R. Gerner, C.-Y. Hsu, M. Catley, R. A. Maciewicz, , L.-F. Lu

This study identifies intestinal regulatory T cells producing IL-27 to specifically regulate Th17 immunity in the gut, revealing a novel mechanism for tissue-specific immune control.

bioRxiv · Feb 20, 2023Immunology

LncRNA Malat1 represses Th17 effector program by maintaining a critical bivalent super-enhancer and promotes intestinal inflammation

S. Ma, B. Zhou, Y. Abe, N. Chen, C. Luo, Y. Li, P. R. Patel, A. Zheng, S. A. Patel, Y. Hao, J. T. Chang, X. Fu, W. J. M. Huang

This study shows that lncRNA Malat1 restricts Th17 cell IL-17A production by regulating a bivalent super-enhancer, thereby controlling intestinal inflammation resolution.

bioRxiv · Mar 21, 2022 · PreprintImmunology

A novel sialic acid-binding adhesin present in multiple species contributes to the pathogenesis of infective endocarditis

M. O. Gaytan, A. K. Singh, S. A. Woodiga, S. A. Patel, S.-S. An, A. Vera Ponce de Leon, S. McGrath, A. R. Miller, J. M. Bush, M. van der Linden, V. Magrini, R. K. Wilson, T. Kitten, S. J. King

This study identifies a novel sialic acid-binding adhesin, AsaA, in IE-causing bacteria that contributes to pathogenesis by binding platelets through sialic acid, expanding the known role of sialic acid as a receptor in bacterial-platelet interactions.

bioRxiv · Jul 17, 2020Microbiology

Epigenetic regulator BMI1 promotes fusion-positive rhabdomyosarcoma proliferation and constitutes a novel therapeutic target

C. E. Shields, S. Potlapalli, S. M. Cuya-Smith, S. K. Chappell, D. Chen, D. Martinez, J. Pogoriler, K. S. Rathi, S. A. Patel, J. M. Maris, R. W. Schnepp

This study shows BMI1 promotes FP-RMS proliferation and its inhibition reduces tumor growth and activates the Hippo pathway.

bioRxiv · Apr 18, 2020Cancer Biology