Author

S. Hertegonne

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 2 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

A. S. Zinkernagel· 2 shared C. Vulin· 2 shared F. Andreoni· 2 shared S. D. Brugger· 2 shared S. Mairpady Shambat· 2 shared T. A. Schweizer· 2 shared A. Gomez-Mejia· 1 shared A. Raez· 1 shared

Activity by Year

2021-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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2 shared papersMicrobiology

A. S. Zinkernagel

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

C-di-AMP levels modulate Staphylococcus aureus cell wall thickness as well as virulence and contribute to antibiotic resistance and tolerance

V. Dengler Haunreiter, A. Tarnutzer, J. Baer, M. von Matt, S. Hertegonne, F. Andreoni, C. Vulin, L. Künzi, C. Menzi, P. Kiefer, P. Christen, J. A. Vorholt, A. S. Zinkernagel

This study shows that high c-di-AMP levels in Staphylococcus aureus increase cell wall thickness and antibiotic resistance, while reducing cell size and antioxidant production.

bioRxiv · Apr 18, 2023Microbiology

Group A Streptococcus strains causing meningitis without distinct invasive phenotype

L. Marquardt, F. Andreoni, M. Boumasmoud, T. A. Schweizer, D. Heuberger, E. Parietti, S. Hertegonne, J. Epprecht, D. Mattle, A. Raez, E. Marques-Maggio, R. Schuepbach, B. K. Hasse, S. Mairpady Shambat, S. D. Brugger, A. Zinkernagel

This study compares GAS strains from meningitis and otitis cases, finding no distinct invasive traits associated with meningitis.

bioRxiv · Mar 22, 2023 · PreprintMicrobiology

Blunted Fas signaling favors RIPK1-driven neutrophil necroptosis in critically ill COVID-19 patients

T. A. Schweizer, S. Mairpady Shambat, C. Vulin, S. Hoeller, C. Acevedo, M. Huemer, A. Gomez-Mejia, C.-C. Chang, J. Baum, S. Hertegonne, E. Hitz, D. A. Hofmaenner, P. K. Buehler, H. Moch, R. A. Schuepbach, , A. S. Zinkernagel

The paper shows that in critically ill COVID-19 patients, dysregulated Fas signaling leads to increased RIPK1-driven necroptosis in neutrophils, contributing to disease severity.

bioRxiv · Mar 19, 2021 · PreprintImmunology