Author

T. Najar

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Assay:flow cytometry· 1 Assay:multiome· 1 Assay:proteomics· 1

Frequent Co-Authors

D. R. Littman· 2 shared L. Kroehling· 2 shared A. Grayson· 1 shared A. Hine· 1 shared A. T. Satpathy· 1 shared C. A. Lareau· 1 shared D. Hudesman· 1 shared D. Li· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

D. R. Littman

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Author profile →

2 papers

Recent·Most cited

Microbiota-instructed regulatory T cell differentiation is mediated by a distinct RORγt+ antigen presenting cell subset

R. Kedmi, T. Najar, K. R. Mesa, A. Grayson, L. Kroehling, Y. Hao, S. Hao, M. Pokrovskii, M. Xu, J. Talbot, J. Wang, J. Germino, C. A. Lareau, A. T. Satpathy, M. S. Anderson, , D. R. Littman

This study identifies a distinct RORγt+ antigen presenting cell subset that mediates microbiota-driven regulatory T cell differentiation, revealing a mechanism for maintaining immune tolerance.

bioRxiv · Nov 19, 2021 · PreprintImmunology

Serum Amyloid A Proteins Induce Pathogenic TH17 Cells and Promote Inflammatory Disease

J.-Y. Lee, J. A. Hall, L. Kroehling, L. Wu, T. Najar, H. H. Nguyen, W.-Y. Lin, S. T. Yeung, H. M. Silva, D. Li, A. Hine, P. Loke, D. Hudesman, J. C. Martin, E. Kenigsberg, , D. R. Littman

This study shows serum amyloid A proteins drive pathogenic TH17 cell differentiation and promote inflammatory disease through STAT3-activating cytokines.

bioRxiv · Jun 25, 2019Immunology