Zsuzsanna Callaerts‐Vegh

The paper investigates the interaction between oxytocin and dopamine in social safety learning in mice using the social transmission of food preference paradigm, finding that oxytocin enhances learning only when dopamine signaling is intact.

This study shows that oxytocin enhances trust acquisition in male mice but blocks trust violation learning, while its antagonist has similar effects, with no impact on females.

This study evaluates the effects of trisomy and GTE-EGCG treatment on multiple systems in a Down syndrome mouse model, finding that treatment exacerbates trisomic phenotypes rather than rescuing them.

This study shows that delivering IL2 to the brain in aging mice partially restores molecular signatures of aging and improves spatial learning.

The paper shows that astrocyte calcium dysfunction leads to early network hyperactivity in Alzheimer's Disease, which can be reversed by restoring calcium signaling.

This study identifies miR-132 deficiency as a cause of impaired adult hippocampal neurogenesis in Alzheimer's disease and shows that restoring miR-132 levels can reverse these deficits and memory issues.

This study shows that CYFIP1 haploinsufficiency in mice causes reduced brain functional connectivity and white matter defects, linking it to neuropsychiatric disorders like Autism and Schizophrenia.