Ken C. Q. Nguyen

This study shows rotavirus uses host microvesicles to transmit immature double-layered particles, enabling non-lytic spread and bypassing outer-capsid-dependent entry.

This study shows that lysosome-related organelle genes are necessary for mitochondrial remodeling during bacterial infections in C. elegans and drive downstream infection-response gene expression.

The paper shows that phasmid sensory neurons and caudal glands coexist in a nematode, challenging the idea that they evolved from each other during aquatic-to-terrestrial transitions.

The paper shows that apical spectrin organizes actin filament bundles in C. elegans to pattern cuticle ridges, and its loss disrupts ridge formation and matrix delamination.

This study shows that dauer-specific gap junction rewiring in C. elegans shortens avoidance duration while preserving initiation, revealing how circuit reorganization balances flexibility and stability under stress.

This study shows that the ECM gene mec-9 affects sensory cilia function and EV shedding in C. elegans through non-autonomous mechanisms.

The paper analyzes ultrastructural data from C. elegans neurons to assess mitochondrial distribution relative to synapses, finding limited sub-cellular control over mitochondrial placement in fine caliber neurons.

The paper studies mitochondrial structural diversity in C. elegans neuromuscular system across development using 3D electron microscopy and deep learning, finding correlations between mitochondrial properties and synaptic connections, and identifying stage-specific mitochondrial features in dauer.

This study shows NEKL-4 regulates ciliary stability and mitochondrial health in C. elegans ciliated neurons through kinesin activation and mitochondrial homeostasis.

The paper reconstructs the dauer connectome of nematodes to show how structural and connectivity changes in neurons lead to specific behaviors during developmental plasticity.

The paper investigates the fate of exophers in C. elegans, showing they are engulfed and degraded by hypodermal cells through a phagocytic process involving specific molecular factors.

The paper shows how actin and a provisional matrix work together to shape acellular ridges in C. elegans cuticle through transient actomyosin constriction and matrix patterning.

The paper shows that gap-junctional signaling via innexins controls the positioning of distal somatic cells in C. elegans gonads without affecting germline stem cell boundaries.

The paper shows that kinesin-3 delivers neurexin to presynaptic terminals, stabilizing dendritic spines and postsynaptic components in C. elegans motor neurons.

The paper shows that Hsp90 chaperones and the F-box protein MEC-15 have opposing roles in neuronal differentiation, with Hsp90 inhibiting neurite growth and synaptic development in C. elegans.

The paper identifies marginal cells in C. elegans pharynx as a sink for hydrophobic small molecules through sphingomyelin synthesis and cholesterol absorption.