Alexandre G. Dayer

This study shows that Htr3a receptors are essential for normal fear response attenuation by regulating oscillatory dynamics and synchronization in the corticolimbic network.

This study shows neurogliaform cells mediate interhemispheric inhibition and modulate sensory perception by affecting pyramidal neuron activity in the mouse barrel cortex.

This study shows that LPA16:0, derived from platelets, reduces adult neurogenesis and stress resilience in anxiety disorders by activating LPA1 receptors.

This study uses high-density EEG to examine eye-gaze processing in adults with ADHD, finding neural abnormalities in attention-sensitive ERP components.

The paper shows that PlexinA4-Semaphorin3A signaling between deep and superficial cortical interneurons regulates their laminar positioning in the developing neocortex.

This study examines how neurofeedback affects alpha oscillations and inhibitory control in adult ADHD patients, finding that post-neurofeedback alpha normalization correlates with improved motor inhibition.

The paper identifies a mutation in the MCF2 gene linked to congenital bilateral perisylvian syndrome and shows it impairs cortical neuronal migration in mice.

This study examines resting-state EEG microstate dynamics in euthymic bipolar disorder patients and finds increased occurrence and coverage of microstate A compared to healthy controls.

The paper shows that polygenic scores for depression traits predict lithium response in bipolar disorder patients, with lower genetic load linked to better treatment outcomes.

The paper traces transcriptional trajectories of progenitors and neurons during mouse neocortex development, showing how molecular states from progenitors shape neuronal diversity.

The paper develops ConnectID, a method linking connectome and transcriptome in single neurons, identifying transcriptional modules associated with axonal projections to specific cortical and subcortical targets.

This study finds that a higher genetic risk for schizophrenia is linked to poorer lithium response in bipolar disorder patients, suggesting potential for personalized treatment strategies.