Alexey V. Semyanov

The paper examines population calcium responses in cortical astrocytes during locomotion, identifying four phases and a temporal threshold of ~5 seconds for superevent formation.

This study shows that increasing ezrin in astrocytes makes mice more resilient to stress-induced depressive behaviors, while decreasing it increases susceptibility.

The paper investigates how GABAergic inputs influence dendritic integration through spatiotemporal differences in polarization and shunting in rat hippocampal neurons.

This study examines how cerebral blood flow and metabolic responses of astrocytes and neurons change during locomotion, revealing distinct mitochondrial behaviors and potential signaling roles of astrocytes.

This study shows electroacupuncture prevents astrocyte atrophy in mice with depression-like behaviors, suggesting astrocytes as a target for treatment.

This study shows that aging impairs astrocyte morphology and mitochondrial function in the human cerebral cortex, while neurons remain unaffected.

The paper shows astrocytes produce nitric oxide from nitrite via mitochondrial sulfite oxidase during hypoxia, modulating cerebral blood flow.

The paper shows that postsynaptic NMDA receptor-mediated K+ efflux reduces astrocytic glutamate uptake while enhancing presynaptic glutamate release.

A high-fat diet increases astrocytic lipid content and branching, enhancing glutamate clearance and exploratory behavior in young mice.

This study shows that ECM attenuation increases synapse number but suppresses synaptic plasticity by reducing neuronal excitability and impairing LTP.

This study shows that aging reduces astrocytic processes and function, which correlates with impaired synaptic plasticity in the hippocampus.

The paper shows circadian rhythms in hippocampal CA1 neurons and astrocytes affect synaptic plasticity through changes in NMDA receptor expression and astrocytic proximity to synapses, altering glutamate clearance and cognitive processing.

This study investigates how tonic GABAA conductance influences different forms of synaptic plasticity, showing distinct effects on spike-timing-dependent and rate-based LTP in rat hippocampal neurons.

This study shows that caloric restriction in mice alters astrocytic morphology and Ca2+ activity, enhancing synaptic plasticity and LTP in hippocampal synapses.

The paper identifies that thin astrocytic processes with high surface-to-volume ratios generate more spontaneous Ca2+ events due to increased baseline fluctuations and CICR.