Author

Marina I. Oliveira da Silva

Universidade do PortoORCID 0000-0002-8941-5805Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cell-cell communication· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Márcia A. LiziD· 3 shared Ricardo Jorge Ferreira TaipaiD· 2 shared Ana Isabel SeixasiD· 1 shared Ana MagalhãesiD· 1 shared Andreia DiasiD· 1 shared António J. FigueiraiD· 1 shared Carolina LemosiD· 1 shared Christiane Fahlbusch· 1 shared

Activity by Year

2024-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersNeuroscience

Márcia A. LizORCID

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3 papers

Recent·Most cited

Rac1 inhibition prevents axonal cytoskeleton dysfunction in Transthyretin Amyloid Polyneuropathy

Joana Magalhães, Vítor Pacheco Dias, Jessica Eira, Marina I. Oliveira da Silva, Guilherme Nóvoa, Margarida Martins, Hung‐Wei Kan, Andreia Dias, Estefânia Carvalho, Mariana Santos, Ana Isabel Seixas, Teresa Coelho, Ricardo Jorge Ferreira Taipa, Joana Paes de Faria, Carolina Lemos, , Márcia A. Liz

This study shows that Rac1 inhibition prevents cytoskeletal dysfunction in ATTRv-PN by rescuing axonal defects in mice and supporting its therapeutic potential in patients.

Cell Reports · Oct 1, 2025Peer-reviewedNeuroscience

Neuronal expression of S100B triggered by oligomeric Aβ peptide protects against cytoskeletal damage and synaptic loss

Joana Saavedra, Mariana Nascimento, António J. Figueira, Marina I. Oliveira da Silva, Tiago Gião, João Filipe Oliveira, Márcia A. Liz, Cláudio M. Gomes, Isabel Cardoso

This study shows that neuronal S100B expression is triggered by Aβ oligomers and protects against cytoskeletal damage and synaptic loss in Alzheimer's disease.

bioRxiv · Apr 4, 2025 · PreprintNeuroscience

Cofilin pathology is a new player on α-synuclein-induced spine impairment in models of hippocampal synucleinopathy

Marina I. Oliveira da Silva, Miguel Santejo, Isaac William Babcock, Ana Magalhães, Laurie S. Minamide, Seok-Joon Won, Erika Castillo, Ellen Gerhardt, Christiane Fahlbusch, Raymond A. Swanson, Tiago F. Outeiro, Ricardo Jorge Ferreira Taipa, Michael R. Ruff, James R. Bamburg, Márcia A. Liz

This study identifies cofilin dysregulation as a mediator of α-synuclein-induced synaptic dysfunction in hippocampal neurons in synucleinopathy models.

Cell Death and Disease · Apr 13, 2024Peer-reviewedNeuroscience