Theodore H. Schwartz

The paper shows that suppressing cortical glutamatergic neurons can paradoxically cause seizures and interictal discharges in vivo.

This study shows that functional connectivity, not anatomical connections, determines seizure spread in a rodent epilepsy model and highlights how stimulation parameters affect neural recruitment.

This study identifies FGF13's role in seizure susceptibility through interneuron-specific knockout mice, revealing a sodium channel-independent mechanism involving K+ channel regulation.

In a mouse model of focal epilepsy, sub-surface laser-produced cuts encircling the seizure focus attenuate propagation without behavioral impairment.

The paper identifies seizure initiation zones in a zebrafish model, showing hyperexcitation in excitatory neurons precedes seizure propagation.

The paper shows that temperature increases in anesthetized rat brains during activation can confound BOLD fMRI signals by being linked to hemoglobin changes.

The paper investigates how inhibitory control affects the spread of focal ictal activity in a rodent model, showing that localized inhibition defects can lead to both classical and epileptic network propagation.

The paper investigates how seizure epicenter depth and laminar synchrony affect tissue oxygenation during ictal initiation in a rat model, revealing layer-dependent hypoxia and hyperoxia patterns.