Author

E. Cantu

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Bioengineering· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 5 Research Focus:cell type annotation· 4 Organism:mouse· 4 Assay:bulk RNA-seq· 3 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 2 Organism:human· 2

Frequent Co-Authors

M. C. Basil· 5 shared J. D. Planer· 3 shared A. E. Vaughan· 2 shared D. B. Frank· 2 shared E. E. Morrisey· 2 shared J. M. Diamond· 2 shared M. P. Morley· 2 shared S. Li· 2 shared

Activity by Year

2023-2024 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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5 shared papersCell Biology

M. C. Basil

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Marginated neutrophils in the lungs effectively compete for nanoparticles targeted to the endothelium, serving as a part of the reticuloendothelial system

M. E. Zamora, E.-O. Essien, K. Bhamidipati, A. Murthy, J. Liu, H. Kim, M. N. Patel, J. Nong, Z. Wang, C. Espy, F. Chaudhry, L. T. Ferguson, S. Tiwari, E. Hood, O. A. Marcos-Contreras, , J. S. Brenner

The paper shows that lung marginated neutrophils effectively uptake endothelial-targeted nanoparticles, suggesting they are part of the reticuloendothelial system.

bioRxiv · Jun 7, 2024Bioengineering

A longitudinal atlas of post-viral lung regeneration reveals persistent injury-associated cell states

T. K. Niethamer, J. D. Planer, M. P. Morley, A. Babu, M. C. Basil, E. Cantu, D. B. Frank, J. M. Diamond, A. N. Nottingham, S. Li, L. I. Levin, S. Zhou, E. E. Morrisey

This study maps the temporal sequence of mouse lung regeneration after viral injury, identifying persistent injury-associated cell states and their role in alveolar niche repair.

bioRxiv · May 24, 2024 · PreprintCell Biology

An injury-induced tissue niche shaped by mesenchymal plasticity coordinates the regenerative and disease response in the lung

D. L. Jones, M. P. Morley, X. Li, Y. Yun, F. L. Cardenas-Diaz, S. Li, S. Zhou, S. E. Schaefer, U. V. Chembazhi, A. Nottingham, S. Lin, E. Cantu, J. M. Diamond, M. C. Basil, A. E. Vaughan, E. E. Morrisey

The paper identifies an injury-induced tissue niche (iTCH) in the lung that coordinates regenerative and disease responses through mesenchymal plasticity and signaling pathways.

bioRxiv · Feb 26, 2024 · PreprintCell Biology

Dysregulated alveolar epithelial cell progenitor function and identity in Hermansky-Pudlak syndrome

J. Y. Wang, N. S. Michki, S. Sitaraman, B. J. Banaschewski, R. Jamal, J. J. Gokey, S. M. Lin, J. B. Katzen, M. C. Basil, E. Cantu, J. A. Kropski, J. A. Zepp, D. B. Frank, L. R. Young

This study identifies intrinsic AT2 progenitor dysfunction and p53 pathway dysregulation as novel mechanisms driving pulmonary fibrosis in Hermansky-Pudlak syndrome.

bioRxiv · Jun 17, 2023Cell Biology

Vascular Endothelial-derived SPARCL1 Exacerbates Viral Pneumonia Through Pro-Inflammatory Macrophage Activation

G. Zhao, M. E. Gentile, L. Xue, C. V. Cosgriff, A. I. Weiner, S. Adams-Tzivelekidis, J. Wong, X. Li, S. Kass-Gergi, N. P. Holcomb, M. C. Basil, K. M. Steward, J. D. Planer, E. Cantu, J. D. Christi, , A. E. Vaughan

This study identifies SPARCL1 as a pro-inflammatory mediator secreted by endothelial cells that worsens viral pneumonia by promoting macrophage activation and is elevated in severe COVID-19 cases.

bioRxiv · May 25, 2023Immunology