Author

Joshua Hartmann

Heidelberg University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Molecular Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

Johannes BacksiD· 3 shared Matthias DewenteriD· 3 shared Almut SchulzeiD· 2 shared Christoph DieterichiD· 2 shared Thiago Britto‐BorgesiD· 2 shared Alireza SaadatmandiD· 1 shared Carsten StichtiD· 1 shared Christian U. OeingiD· 1 shared

Activity by Year

2025-2025 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersCell Biology

Johannes BacksORCID

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3 papers

Recent·Most cited

Genomic G quadruplexes regulate mRNA splicing

Victoria Mauz, Thiago Britto‐Borges, Tobias S. Merkel, Enio Gjerga, Joshua Hartmann, Marteinn Thor Snaebjornsson, Friederike C. Schreiter, Christian U. Oeing, Carsten Sticht, Samuel Tobias Sossalla, Jürgen Günther Okun, Georg Stoecklin, Matthias W. Hentze, Eileen E. M. Furlong, Almut Schulze, , Johannes Backs

The paper shows that genomic G quadruplexes regulate mRNA splicing by recruiting RNA-binding proteins, with implications for cardiac function.

bioRxiv · Dec 16, 2025 · PreprintCell Biology

CAMK2-NR4A1 signaling initiates metabolic substrate switching to induce heart failure with reduced ejection fraction

Alireza Saadatmand, Mark E. Pepin, Zihao Chen, Joshua Hartmann, Qiang Sun, Matthias Dewenter, Sumra Nazir, Harikrishnareddy Paluvai, Marco Hagenmüller, Uwe Haberkorn, Lisa Schlicker, Roberto Carlos Frias-Soler, Jens Tyedmers, Christoph Maack, Almut Schulze, Johannes Backs

The paper identifies CAMK2-NR4A1 signaling as a driver of metabolic substrate switching in heart failure with reduced ejection fraction, linking it to cardiac dysfunction through glucose utilization and lipid depletion.

bioRxiv · Oct 16, 2025 · PreprintPhysiology

CAMK2D causes heart failure in RBM20 cardiomyopathy

Maarten M.G. van den Hoogenhof, Javier Durán, Thiago Britto‐Borges, Elena Kemmling, Laura Konrad, David C. Lennermann, Joshua Hartmann, Laura Schraft, Julia Kornienko, Theresa Bock, Marcus Krüger, Christoph Dieterich, Lars Michael Steinmetz, Matthias Dewenter, Johannes Backs

This study identifies CAMK2D overactivation as the cause of heart failure in RBM20 cardiomyopathy and shows pharmacological inhibition can reverse cardiac dysfunction.

bioRxiv · May 23, 2025 · PreprintMolecular Biology