Author

Jowana Obeid

Stony Brook Medicine

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Approach:biophysical modeling· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Mehdi DamaghiiD· 3 shared Andrew E ResnickiD· 2 shared Christopher J. ClarkeiD· 2 shared Fabiola Noelia VelazqueziD· 2 shared Joon-Hyun SongiD· 2 shared Yusuf Awni HannuniD· 2 shared A Chen· 1 shared Adriana Del Pino Herrera· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersCancer Biology

Mehdi DamaghiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Neutral Sphingomyelinase-2 Restrains TAZ to Suppress Breast Tumor Growth

Andrew E Resnick, Victoria Franzi, Botheina Ghandour, Sam B Chiappone, Sébastien Lalanne, Monica Alexander, Deanna M Peperno, Jowana Obeid, Ishmam Pritam, Ian D Miranda, Nicolas Coant, Michael V. Airola, Mehdi Damaghi, Fabiola Noelia Velazquez, Yusuf Awni Hannun, Christopher J. Clarke

This study identifies nSMase2 as a metabolic tumor suppressor in breast cancer by restraining TAZ and inhibiting tumorigenesis.

bioRxiv · Jul 4, 2026 · PreprintCancer Biology

Metabolic plasticity enables cancer adaptation to acidic tumor ecosystems

Raafat Chalar, Naheel Khatri, Jowana Obeid, Joon-Hyun Song, Yujie Xiao, Saiful Samad, A Chen, Andrew E Resnick, K H Karbalaei, Janet J. Allopenna, Cungui Mao, Christopher J. Clarke, Fabiola Noelia Velazquez, Bo Chen, Daniel Canals, , Mehdi Damaghi

This study shows cancer cells adapt to acidic tumor environments through sphingolipid metabolic plasticity, revealing that targeting metabolic flexibility, not individual pathways, may be more effective.

bioRxiv · May 5, 2026 · PreprintCancer Biology

Metabolic plasticity increases evolvability and drives persistence to PARP inhibitor in ovarian cancer

Adriana Del Pino Herrera, Joon-Hyun Song, Karla Torres‐Arciga, Susoma Halder, Kadin El Bakkouri, Jowana Obeid, William M. Burke, Mehdi Damaghi, Meghan C. Ferrall-Fairbanks

Ovarian cancer cells exploit metabolic plasticity to increase their evolvability to persist PARP inhibitor treatment.

bioRxiv · Oct 8, 2025 · PreprintCancer Biology