Author

K. A. Raymond

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 4 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 4 Organism:human· 4 Assay:bulk RNA-seq· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1

Frequent Co-Authors

S. K. Pillai· 4 shared H. S. Sperber· 3 shared M. S. Bouzidi· 3 shared A. Cruz· 1 shared A. G. Gutierrez· 1 shared A. Vallejo-Gracia· 1 shared B. Korber· 1 shared D. Boehm· 1 shared

Activity by Year

2020-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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4 shared papersMicrobiology

S. K. Pillai

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

APOBEC3F is the main source of editing identified during the 2022 outbreak of human monkeypox virus

R. Suspene, K. A. Raymond, L. Boutin, S. Guillier, F. Lemoine, O. Ferraris, J.-N. Tournier, F. Iseni, E. Simon-Loriere, J.-P. Vartanian

The paper identifies APOBEC3F as the main source of hypermutation in MPXV during the 2022 outbreak, showing its role in generating G->A mutations and subsequent viral evolution.

bioRxiv · Jan 6, 2023 · PreprintMicrobiology

The Hypoxia-regulated Ectonucleotidase CD73 is a Host Determinant of HIV Latency

H. S. Sperber, K. A. Raymond, M. S. Bouzidi, T. Ma, S. Valdebenito, E. Eugenin, N. R. Roan, S. G. Deeks, S. Winning, J. Fandrey, R. Schwarzer, S. K. Pillai

This study identifies CD73 as a marker of HIV latency, showing hypoxia enhances its expression and adenosine signaling promotes viral quiescence.

bioRxiv · Aug 3, 2022Microbiology

μ-Lat: A Mouse Model to Evaluate Human Immunodeficiency Virus Eradication Strategies

H. S. Sperber, P. P. Togarrati, K. A. Raymond, M. S. Bouzidi, R. Gilfanova, A. G. Gutierrez, M. O. Muench, S. K. Pillai

The paper develops a humanized mouse model to evaluate HIV eradication strategies by transplanting J-Lat cells and testing TNF's effect on viral reactivation.

The FASEB Journal · Sep 9, 2020Peer-reviewedMicrobiology

Durable Control of HIV-1 Using a Staphylococcus aureus Cas9-Expressing Lentivirus Co-Targeting Viral Latency and Host Susceptibility

L. R. Chavez, N. R. Reddy, K. A. Raymond, M. S. Bouzidi, S. Desai, Z. Y. Dossani, H. S. Sperber, J. Theiler, B. Korber, S. K. Pillai

This study develops a lentiviral vector targeting both HIV-1 provirus and host CXCR4 to reduce gene editing efficacy thresholds for an HIV-1 cure.

bioRxiv · Aug 10, 2020 · PreprintMicrobiology

FOXO1 promotes HIV Latency by suppressing ER stress in T cells

A. Vallejo-Gracia, I. P. Chen, R. Perrone, E. Besnard, D. Boehm, E. Battivelli, T. Tezil, K. Krey, K. A. Raymond, P. A. Hull, M. Walter, I. Habrylo, A. Cruz, S. Deeks, S. K. Pillai, , M. Ott

This study shows FOXO1 promotes HIV latency by suppressing ER stress in T cells and identifies pathways to selectively reverse quiescence and reduce viral reservoirs.

bioRxiv · Apr 23, 2020Immunology