Author

M. A. Aziz

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3

Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Organism:chicken / avian· 1 Approach:statistical model· 1

Frequent Co-Authors

E. Sugrue· 2 shared J. Hughes· 2 shared M. Palmarini· 2 shared M. Varela· 2 shared S. J. Rihn· 2 shared S. J. Wilson· 2 shared A. da Silva Filipe· 1 shared A. Filipe· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 28 mutual co-author links

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2 shared papersMicrobiology

E. Sugrue

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Timing and magnitude of the type-I interferon response are determinants of disease tolerance in arbovirus infection

A. Hardy, S. Bakshi, W. Furnon, O. MacLean, Q. Gu, M. Varjak, M. Varela, M. A. Aziz, A. Shaw, R. M. Pinto, N. C. Ruiz, C. Mullan, A. Taggart, A. Filipe, R. E. Randall, , M. Palmarini

This study shows that the timing and magnitude of the type-I interferon response determine disease tolerance in bluetongue virus infection between sheep and cattle.

bioRxiv · Oct 19, 2022 · PreprintMicrobiology

The Interferon Resistance of Transmitted HIV-1 is a Consequence of Enhanced Replicative Fitness

E. Sugrue, A. Wickenhagen, N. Mollentze, M. A. Aziz, V. B. Sreenu, S. Truxa, L. Tong, A. da Silva Filipe, D. L. Robertson, J. Hughes, S. J. Rihn, S. J. Wilson

The paper identifies that transmitted HIV-1's interferon resistance stems from enhanced replicative fitness rather than specific IFN resistance.

bioRxiv · Dec 18, 2021Microbiology

TRIM69 inhibits Vesicular Stomatitis Indiana Virus (VSIV)

S. J. Rihn, M. A. Aziz, D. G. Stewart, J. Hughes, M. L. Turnbull, M. Varela, E. Sugrue, C. S. Herd, M. Stanifer, S. P. Sinkins, M. Palmarini, S. J. Wilson

This study identifies TRIM69 as an interferon-induced gene that potently inhibits VSIV, with specific antiviral activity and evolutionary signatures of positive selection.

bioRxiv · Jun 13, 2019Microbiology