Author

M. Horvath

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Biochemistry· 1 Bioengineering· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 4 Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:host-pathogen & immune response· 2 Approach:biophysical modeling· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1

Frequent Co-Authors

A. Gacser· 2 shared C. Vagvolgyi· 2 shared D. C. Castaneda· 2 shared E. Fleming· 2 shared J. Oh· 2 shared K. Palucka· 2 shared M. Callender· 2 shared P. Horvath· 2 shared

Activity by Year

2021-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersMicrobiology

A. Gacser

Mutual co-authors in cluster

Author profile →

5 papers

Recent·Most cited

Species- and strain-effects in interferon, innate immunity, and barrier transcriptional response of microbiome interactions of 3D respiratory epithelial cultures

M. Horvath, R. Yang, D. C. Castaneda, M. Callender, E. Fleming, A. Voigt, R. Caldwell, J. Fachi, B. Di Luccia, Z. Scholar, P. Yu, A. Salner, M. Colonna, K. Palucka, J. Oh

The paper examines how different microbes affect host immune and barrier responses in respiratory epithelial cultures, finding that interferon variability is not linked to microbial diversity or other innate pathways.

bioRxiv · Sep 25, 2023 · PreprintMicrobiology

High-Throughput Bioprinting of the Nasal Epithelium using Patient-derived Nasal Epithelial Cells

I. D. Derman, M. Yeo, D. C. Castaneda, M. Callender, M. Horvath, Z. Mo, R. Xiong, E. Fleming, P. Chen, M. E. Peeples, K. Palucka, J. Oh, I. T. Ozbolat

This study uses 3D bioprinting to create nasal epithelium models from patient-derived cells, enabling high-throughput fabrication and recapitulating native epithelial architecture and virus infection capability.

bioRxiv · Mar 29, 2023Bioengineering

O-GlcNAc modification forces the formation of an α-Synuclein amyloid-strain with notably diminished seeding activity and pathology

A. T. Balana, A.-L. Mahul-Mellier, B. A. Nguyen, M. Horvath, A. Javed, E. R. Hard, Y. Jasiqi, P. Singh, S. Afrin, R. Pedretti, V. Singh, V. M.-Y. Lee, K. C. Luk, L. Saelices, H. A. Lashuel, M. R. Pratt

This study shows that O-GlcNAc modification of α-Synuclein leads to amyloid fibrils with reduced seeding activity and pathology in Parkinson's disease models.

bioRxiv · Mar 7, 2023 · PreprintBiochemistry

Candida albicans enhances the progression of oral squamous cell cancrinoma in vitro and in vivo

M. Vadovics, N. Igaz, R. Alfoldi, D. Rakk, E. Veres, B. Szucs, M. Horvath, R. Toth, A. Szucs, P. Horvath, L. Tiszlavicz, C. Vagvolgyi, J. Nosanchuk, A. Szekeres, M. Kiricsi, , A. Gacser

This study shows that C. albicans promotes OSCC progression in vitro and in vivo by inducing inflammation, oncometabolites, and metastatic gene expression.

bioRxiv · Mar 31, 2021 · PreprintMicrobiology

Oral epithelial cells distinguish between Candida species with high or low pathogenic potential through miRNA regulation

M. Horvath, G. Nagy, N. Zsindely, L. Bodai, P. Horvath, C. Vagvolgyi, J. D. Nosanchuk, R. Toth, A. Gacser

The paper shows oral epithelial cells respond differently to C. albicans and C. parapsilosis, with miRNA regulation playing a key role in distinguishing their pathogenic potential.

bioRxiv · Jan 23, 2021 · PreprintMicrobiology