Author

M.-L. Thezenas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Assay:proteomics· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:host-pathogen & immune response· 2 Research Focus:development & morphogenesis· 1 Organism:Drosophila· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

A. Simmons· 2 shared B. M. Kessler· 2 shared D. Corridoni· 2 shared R. Fischer· 2 shared S. Shiraishi· 2 shared T. Chapman· 2 shared A. Aulicino· 1 shared B. R. Hopkins· 1 shared

Activity by Year

2019-2019 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersImmunology

A. Simmons

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Ejaculate deterioration with male age, and its amelioration in Drosophila

I. Sepil, B. R. Hopkins, R. Dean, E. Bath, S. Friedman, B. Swanson, H. J. Ostridge, N. A. Buehner, M. F. Wolfner, R. Konietzny, M.-L. Thezenas, E. Sandham, P. D. Charles, R. Fischer, J. Steinhauer, , S. Wigby

The paper shows ejaculate performance declines with male age in Drosophila due to sperm and seminal protein deterioration, but this decline can be ameliorated by targeting insulin-like peptide-producing cells.

bioRxiv · Jun 28, 2019 · PreprintEvolutionary Biology

Ataxin-3 links NOD2 and TLR2 mediated innate immune sensing and metabolism in myeloid cells

T. Chapman, D. Corridoni, S. Shiraishi, S. Pandey, A. Aulicino, S. Wigfield, M. do Carmo Costa, M.-L. Thezenas, H. Paulson, R. Fischer, B. M. Kessler, A. Simmons

This study identifies ataxin-3 as a mediator of NOD2 and TLR2 signaling in myeloid cells, linking innate immune sensing to metabolic processes and mitochondrial function.

bioRxiv · Apr 1, 2019Immunology

NOD2 and TLR2 Signal via TBK1 and PI31 to Direct Cross-presentation and CD8 T Cell Responses

D. Corridoni, S. Shiraishi, T. Chapman, T. Steevels, D. Muraro, M.-L. Thezenas, G. Prota, J.-L. Chen, U. Gileadi, N. Ternette, V. Cerundolo, A. Simmons

This study identifies TBK1 and PI31 as key mediators of NOD2 and TLR2 signaling in dendritic cells, enhancing cross-presentation and CD8+ T cell responses against bacterial pathogens.

bioRxiv · Mar 8, 2019Immunology