Author

Evangelia Malahias

Calico Life SciencesORCID 0000-0002-3147-153XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Immunology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Aaron W. StebbinsiD· 2 shared Chunlian ZhangiD· 2 shared David G. HendricksoniD· 2 shared James J. LeeiD· 2 shared Jose Zavala‐SolorioiD· 2 shared Patrick M. GodfreyiD· 2 shared Po-Han TaiiD· 2 shared Varahram ShahryariiD· 2 shared

Activity by Year

2022-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersCell Biology

Aaron W. StebbinsORCID

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3 papers

Recent·Most cited

Wnt signaling decline drives age-related alveolar stem cell loss and impairs lung repair

Tsotne Chitiashvili, Alexander L. Li, Agnieszka A. Wendorff, K. Sivasubramanian, Wenjun Kong, Erik Arroyo-Colon, Ziyou Ren, Evangelia Malahias, Po-Han Tai, Georgina Duenas, John C. K. Wang, Kexin Kong, Ngoc Vu, John M. Patino, Wendy Craft, , Astrid Gillich

This study identifies declining Wnt signaling as a driver of age-related alveolar stem cell loss and shows that restoring Wnt signaling in aged mice improves lung repair but not full regeneration.

bioRxiv · Aug 21, 2026 · PreprintCell Biology

PTPN1/2 inhibits alveolar macrophage-mediated control of lung metastasis

Yue Liu, Im-Meng Sun, Marc Creixell, Jordan Brown, Samir Kharbanda, James J. Lee, Varahram Shahryari, Kayley Hake, Jacqueline O'Hara, Kenneth J. Finn, Nianxin Yang, Lolita Penland, Jiaxi Wang, Ka Man Li, John Balibalos, , Jonathan D. Powell

This study shows that inhibiting PTPN1/2 enhances alveolar macrophage anti-metastatic activity through IFN-gamma-STAT1 signaling, offering a therapeutic strategy for metastasis.

bioRxiv · Feb 26, 2026 · PreprintImmunology

Partial reprogramming restores youthful gene expression through transient suppression of cell identity

Antoine E. Roux, Chunlian Zhang, Jonathan S. Paw, Jose Zavala‐Solorio, Evangelia Malahias, Twaritha Vijay, Ganesh A. Kolumam, Cynthia J. Kenyon, Jacob C. Kimmel

This study investigates how transient reprogramming affects somatic cell identity and aging in mice, finding that it can restore youthful gene expression but also suppress identity programs.

Cell Systems · Jun 10, 2022Peer-reviewedSystems Biology