Author

Sumana Sanyal

University of OxfordORCID 0000-0002-6230-5366Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:host-pathogen & immune response· 3 Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

Mingyuan LiiD· 2 shared Qi Wen TeoiD· 2 shared Adán Pinto‐Fernández· 1 shared Adrianna Banducci-Karp· 1 shared Anna Beekmayer-DhilloniD· 1 shared Charlotte E. Melia· 1 shared Christopher B. BrookeiD· 1 shared Edward DrydaleiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 19 mutual co-author links

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2 shared papersCell Biology

Mingyuan LiORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

UCHL3 regulates subgenomic flaviviral RNA condensates to promote virus propagation

Óscar Trejo-Cerro, Anna Beekmayer-Dhillon, Qi Wen Teo, Lewis Yu-Lam Siu, Mingyuan Li, Kate J. Heesom, Adán Pinto‐Fernández, Sumana Sanyal

This study identifies UCHL3 as a host factor that stabilizes subgenomic flaviviral RNA condensates, promoting virus replication through interferon-independent mechanisms.

Advanced Science · Jun 3, 2026Peer-reviewedCell Biology

TRIM21 is a molecular rheostat for influenza A virus replication

Qi Wen Teo, Huibin Lv, Tossapol Pholcharee, Xin Chen, Vidia Ramadin, Kevin J. Mao, Jessica J. Huang, Joel Rivera-Cardona, Jie Zhu, Yang Wei Huan, Evan K. Shao, Hein Min Tun, Christopher B. Brooke, Lilliana Radoshevich, Sumana Sanyal, Nicholas C. Wu

This study shows TRIM21 acts as a molecular rheostat regulating influenza A virus replication by modulating ubiquitination and innate immune signaling, with PRKDC as a compensatory antiviral factor when TRIM21 is absent.

bioRxiv · Mar 17, 2026 · PreprintCell Biology

Remodelled cholesteryl ester enriched lipid droplets fuel flavivirus morphogenesis

Adrianna Banducci-Karp, Sophie Brixton, Pranav N. M. Shah, Mingyuan Li, Georgina Fisher, Joey Riepsaame, Raman Dhaliwal, Katie L. Holden, Edward Drydale, James Bancroft, Charlotte E. Melia, Gathsaurie Neelika Malavige, Sumana Sanyal

This study identifies cholesteryl ester-enriched lipid droplets as critical host components required for flavivirus infection and shows their role in viral morphogenesis.

bioRxiv · Jun 7, 2025 · PreprintCell Biology