Author

Kevin Christian Montecillo Gulay

University of California San DiegoORCID 0000-0002-9568-3590Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 4

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:drug response / drug discovery· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Hervé TiriaciD· 3 shared Alexei MartsinkovskiyiD· 2 shared Andrew M. LowyiD· 2 shared Evangeline Sari. MoseiD· 2 shared Jay PateliD· 2 shared Aimee L. EdingeriD· 1 shared Alexander KlegeriD· 1 shared Alexander MuiriD· 1 shared

Activity by Year

2025-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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3 shared papersCancer Biology

Hervé TiriacORCID

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4 papers

Recent·Most cited

Redirecting cytomegalovirus immunity against pancreas cancer for immunotherapy

Rémi Marrocco, Jay Patel, Rithika Medari, Philip Salu, Eduardo Lucero-Meza, Catarina Maia, Simon Brunel, Alexei Martsinkovskiy, Siming Sun, Kevin Christian Montecillo Gulay, Malak Jaljuli, Evangeline Sari. Mose, Andrew Lowy, Chris A. Benedict, Tatiana Hurtado de Mendoza

This paper redirects cytomegalovirus (CMV)-specific T cells to target pancreatic cancer, showing improved tumor control and survival in mice.

Journal for ImmunoTherapy of Cancer · Feb 1, 2026Peer-reviewedCancer Biology

Mevalonate Biosynthesis is a Metabolic Vulnerability of Gemcitabine-resistant Pancreatic Cancer

Alica K. Beutel, Sabrina E. Calderon, Ethan Nghiem, Kevin Christian Montecillo Gulay, Leonor Patricia Schubert Santana, Eleni Zimmer, Rima Singh, Cecily Anaraki, Natalie Yousefian, Chantal Allgöwer, Gregory Tong, Cameron Geller, Alina Chao, Dennis Juarez, Thomas Theodor Werner Seufferlein, , Christopher J. Halbrook

This study identifies mevalonate biosynthesis as a therapeutic target and biomarker for gemcitabine-resistant pancreatic cancer.

bioRxiv · Sep 19, 2025 · PreprintCancer Biology

ZBTB11 Depletion Targets Metabolic Vulnerabilities in K-Ras Inhibitor Resistant PDAC

Nathan L. Tran, Jiewei Jiang, Mengyao Ma, Gillian E. Gadbois, Kevin Christian Montecillo Gulay, Alyssa L. Verano, Cara R. Schiavon, Elena Rebollo, Haowen Zhou, Chun‐Teng Huang, Rabi Murad, David A. Scott, Uri Manor, Anne G. Bang, Hervé Tiriac, , Fleur M. Ferguson

This study develops ZBTB11 degraders to target metabolic vulnerabilities in K-Ras inhibitor-resistant PDAC, showing improved anti-cancer activity with reduced toxicity to neurons.

Nature Chemical Biology · Aug 11, 2025Peer-reviewedCancer Biology

MICAL2 Is a Super Enhancer Associated Gene that Promotes Pancreatic Cancer Growth and Metastasis

Bharti Garg, Sohini Khan, Asimina S. Courelli, Ponmathi Panneerpandian, Deepa Sheik Pran Babu, Evangeline Sari. Mose, Kevin Christian Montecillo Gulay, Shweta Sharma, Divya Sood, Alexander T. Wenzel, Alexei Martsinkovskiy, Nirakar Rajbhandari, Jay Patel, Dawn Jaquish, Edgar Esparza, , Andrew M. Lowy

This study identifies MICAL2 as a super enhancer-associated gene that promotes pancreatic cancer growth and metastasis through multiple molecular mechanisms.

Cancer Research · Jan 2, 2025Peer-reviewedCancer Biology