Author

A. Woloszynska

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 3

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 3 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:clustering· 1 Research Focus:host-pathogen & immune response· 1 Assay:metabolomics· 1

Frequent Co-Authors

S. Ramakrishnan· 2 shared W. J. Huss· 2 shared A. Gupta· 1 shared B. A. Foster· 1 shared B. K. Farmer· 1 shared B. M. Gillard· 1 shared D. J. Smiraglia· 1 shared E. C. Gomez· 1 shared

Activity by Year

2021-2021 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

S. Ramakrishnan

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

DDK inhibition disrupts replication leading to mitotic catastrophe in Ewing sarcoma

J. C. Martin, A. Gupta, T. J. Hagoel, L. Gao, M. L. Lynch, A. Woloszynska, T. Melendy, J. Kane, J. Kuechle, J. Ohm

This study shows that DDK inhibition causes replication stress and mitotic catastrophe in Ewing sarcoma cells, reducing viability and suggesting DDK as a therapeutic target.

bioRxiv · Nov 2, 2021 · PreprintCancer Biology

Racial differences in androgen metabolism and receptor signaling in prostate cancer

S. Ramakrishnan, E. C. Gomez, K. Attwood, R. A. Kittles, J. Wang, S. R. Rosario, D. J. Smiraglia, G. Azabdaftari, J. L. Mohler, W. J. Huss, A. Woloszynska

This study identifies racial differences in androgen metabolism and receptor signaling in prostate cancer, showing African American men use an alternative pathway for DHT biosynthesis associated with disease progression.

bioRxiv · Mar 31, 2021 · PreprintCancer Biology

Patient derived models of bladder cancer amplify tumor specific gene expression compared to surgical specimen while maintaining gene expression of molecular subtype and epithelial mesenchymal transition markers

M. Mastri, S. Ramakrishnan, S. Shah, E. Karasik, B. M. Gillard, M. M. Moser, B. K. Farmer, G. S. Chatta, A. Woloszynska, K. H. Eng, B. A. Foster, W. J. HUSS

This study compares gene expression profiles of patient-derived models (PDMs) with surgical specimens of bladder cancer, finding PDXs most similar to original tumors while maintaining EMT and molecular subtype markers.

bioRxiv · Feb 10, 2021 · PreprintCancer Biology