Author

Julie Allen

University of Kansas Medical Center

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3 Biochemistry· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 5 Approach:experimental assay· 4 Assay:proteomics· 4 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Research Focus:host-pathogen & immune response· 1 Research Focus:metabolism & bioenergetics· 1 Approach:statistical model· 1

Frequent Co-Authors

Edziu FranczakiD· 5 shared John P. ThyfaultiD· 5 shared Benjamin A. KugleriD· 4 shared Sebastian F. SalatheiD· 4 shared E. Matthew MorrisiD· 3 shared Peter A. CrawfordiD· 3 shared Colin S. McCoiniD· 2 shared Frederick B. BoakyeiD· 2 shared

Activity by Year

2025-2026 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 44 mutual co-author links

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5 shared papersPhysiology

Edziu FranczakORCID

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Author profile →

5 papers

Recent·Most cited

Neuronal ketone body utilization couples exercise and time-restricted feeding to cognitive enhancement

Sebastian F. Salathe, Benjamin A. Kugler, Edziu Franczak, Xin C. Davis, Frederick B. Boakye, Julie Allen, Kyle Fulghum, Eric D. Queathem, E. Matthew Morris, Patrycja Puchalska, Peter A. Crawford, John P. Thyfault

This study investigates the role of neuronal and hepatic ketone metabolism in cognitive enhancement from exercise and time-restricted feeding in mice, finding neuronal utilization is essential while hepatic production is contributory.

Molecular Metabolism · Jul 16, 2026Peer-reviewedPhysiology

Hepatic ketogenic insufficiency blunts exercise-induced energy expenditure and alters mitochondrial proteins in skeletal muscle.

Xin C. Davis, Colin S. McCoin, Sebastian F. Salathe, Edziu Franczak, Julie Allen, Eric D Queatham, Kyle Fulghum, Patrycja Puchalska, Peter A. Crawford, John P. Thyfault, E. Matthew Morris

This study shows that liver-specific HMGCS2 knockout reduces exercise-induced energy expenditure and alters mitochondrial proteins in skeletal muscle.

Function · May 18, 2026Peer-reviewedPhysiology

Loss of ovarian function and estrogen therapy remodel the brain's synaptic and metabolic proteome

Sebastian F. Salathe, Edziu Franczak, Zane Busick, Frederick B. Boakye, Julie Allen, Andrew J. Lutkewitte, Heather Wilkins, John P. Thyfault, Benjamin A. Kugler

This study shows that estrogen therapy restores synaptic and metabolic proteins in the brain of aged mice after ovariectomy, improving mitochondrial function and synaptic integrity.

American Journal of Physiology-Regulatory, Integrative and Comparative Physiology · Apr 2, 2026Peer-reviewedNeuroscience

Hepatocyte Estrogen Receptor α Mediates Estrogen-induced Augmentation of Hepatic 1 Mitochondrial Respiration Following Ovariectomy.

Edziu Franczak, Benjamin A. Kugler, Sebastian F. Salathe, Julie Allen, Colin S. McCoin, E. Matthew Morris, John P. Thyfault

This study shows that estrogen receptor α in hepatocytes is necessary for estrogen to enhance mitochondrial respiration and reduce steatosis after ovariectomy.

bioRxiv · Feb 23, 2026 · PreprintBiochemistry

Aerobic capacity and exercise mediate protection against hepatic steatosis via enhanced bile acid metabolism.

Benjamin A. Kugler, Adrianna Maurer, Xiaorong Fu, Edziu Franczak, Nicholas J. Ernst, Kevin Schwartze, Julie Allen, Tiangang Li, Peter A. Crawford, Lauren G. Koch, Steven L. Britton, Kartik Shankar, Shawn C. Burgess, John P. Thyfault

The paper shows aerobic capacity and exercise enhance bile acid metabolism, which is critical for reducing hepatic steatosis in mice.

Function · Jan 1, 2025Peer-reviewedPhysiology