Author

Mark Turmaine

University College London

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1 Organism:zebrafish· 1

Frequent Co-Authors

Hugo CabedoiD· 2 shared Jose Antonio Gomez-SancheziD· 2 shared Peter Arthur‐FarrajiD· 2 shared Shaline V. FazaliD· 2 shared Agueda A. RostagnoiD· 1 shared Alastair Morris KilpatrickiD· 1 shared André StrydomiD· 1 shared Andrea LoretoiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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2 shared papersNeuroscience

Hugo CabedoORCID

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Author profile →

3 papers

Recent·Most cited

SARM1 detection in oligodendrocytes but not Schwann cells though sarm1/Sarm1 deletion does not perturb CNS nor PNS myelination in zebrafish and mice.

Shaline V. Fazal, Clara Mutschler, Civia Zi-Yu Chen, Mark Turmaine, Chiung-Ya Chen, Yi‐Ping Hsueh, Andrea Loreto, Ángeles Casillas-Bajo, Hugo Cabedo, Robin J.M. Franklin, Roger A. Barker, Kelly R. Monk, Benjamin Steventon, Michael P. Coleman, Jose Antonio Gomez-Sanchez, Peter Arthur‐Farraj

The paper shows SARM1 is present in oligodendrocytes but not Schwann cells, and its deletion does not affect myelination in zebrafish or mice.

bioRxiv · Dec 8, 2022 · PreprintNeuroscience

Failures of nerve regeneration caused by aging or chronic denervation are rescued by restoring Schwann cell c-Jun

Laura J. Wagstaff, Jose Antonio Gomez-Sanchez, Shaline V. Fazal, Georg Otto, Alastair Morris Kilpatrick, Kirolos G. F. T. Michael, Liam Y.N. Wong, Ki Hwan Ma, Mark Turmaine, John P. Svaren, Tessa Gordon, Peter Arthur‐Farraj, Sergio Velasco-Avilés, Hugo Cabedo, Cristina Benito, , Kristján R. Jessen

The paper shows that restoring Schwann cell c-Jun rescues nerve regeneration failures in aging and chronic denervation by enhancing regenerative support.

eLife · Jan 21, 2021Peer-reviewedNeuroscience

Patient-specific Alzheimer-like pathology in trisomy 21 cerebral organoids reveals BACE2 as a gene-dose-sensitive AD-suppressor in human brain

Ivan Alić, Pollyanna Agnes Goh, Aoife Murray, Erik Portelius, Eleni Gkanatsiou, Gillian Gough, Kin Ying Mok, David Koschut, Reinhard Brunmeir, Yee Jie Yeap, Niamh Louise O’Brien, Jürgen Groet, Xiaowei Shao, Steven Havlicek, Norris Ray Dunn, , Henrik Zetterberg

The paper shows that trisomy 21 cerebral organoids exhibit AD-like pathology, which can be suppressed by BACE2 trisomy and inhibited by BACE1 inhibitors, suggesting BACE2 as an AD-suppressor gene.

Molecular Psychiatry · Jul 9, 2020Peer-reviewedNeuroscience