Author
University of Massachusetts AmherstORCID 0000-0002-4123-0633Unclaimed ORCID profile · follow to get notified of new preprints
5 papers
Recent·Most cited
Adam C. Lynch, Kaito A. Hioki, Xueting Liang, Iris Thesmar, Julia Cernjul, Xin‐Jian He, Jesse Mager, Wei Cui, Dominique Alfandari, Elena L. Pobezinskaya, Leonid A. Pobezinsky
The paper identifies a Dapl1+ subset of naive CD8 T cells that are pre-programmed to differentiate into memory-like T cells.
bioRxiv · Feb 6, 2025 · PreprintImmunology
Feng Wang, Ashmita Chander, Yeonsoo Yoon, Janelle M. Welton, Mary C. Wallingford, Carmen Espejo-Serrano, Francisco Bustos, Greg M. Findlay, Jesse Mager, Ingolf Bach
The paper investigates the role of imprinted XCI in polar trophoblast cells and shows that the Rlim-Rex1 axis is essential for maintaining their fate and preventing early lethality in female mice.
Proceedings of the National Academy of Sciences · Dec 19, 2023Peer-reviewedDevelopmental Biology
Pilar Cacheiro, Carl Henrik Westerberg, Jesse Mager, Mary E. Dickinson, Lauryl M. J. Nutter, Violeta Muñoz‐Fuentes, Chih‐Wei Hsu, Ignatia B. Van den Veyver, Ann Marie Flenniken, Colin McKerlie, Stephen A. Murray, Lydia Teboul, Jason D. Heaney, K. C. Kent Lloyd, Louise Lanoue, , John Pullinger
The paper identifies novel Mendelian genes by analyzing mouse embryo lethality and linking them to human disease phenotypes and genetic variants.
Genome Medicine · Oct 13, 2022Peer-reviewedGenetic & Genomic Medicine
Feng Wang, Maria G. Gervasi, Ana Bošković, Fengyun Sun, Vera D. Rinaldi, Jun Yu, Mary C. Wallingford, Darya A. Tourzani, Jesse Mager, Lihua J. Zhu, Oliver J. Rando, Pablo E. Visconti, Lara Strittmatter, Ingolf Bach
This study shows that Rlim is essential for spermiogenesis in male mice, as its deletion leads to defective sperm production and fertility issues.
eLife · Feb 23, 2021Peer-reviewedDevelopmental Biology
Panpan Zhao, Huanan Wang, Han Wang, Yanna Dang, Lei Luo, Shuang Li, Yan Shi, Lefeng Wang, Shaohua Wang, Jesse Mager, Kun Zhang
This study shows that co-knockdown of Hdac1 and 2 causes developmental failure in mouse embryos due to impaired lineage specification, increased apoptosis, and altered DNA methylation.
Epigenetics · Sep 19, 2019Peer-reviewedDevelopmental Biology