Author

Valentina Baena

National Institutes of HealthORCID 0000-0002-6429-439XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Achim A WerneriD· 1 shared Amy K. WangiD· 1 shared Brecca MilleriD· 1 shared Caroline N. DealyiD· 1 shared Dan Avi LandauiD· 1 shared David B. BeckiD· 1 shared Deborah Kaback· 1 shared Deborah Rivera· 1 shared

Activity by Year

2017-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperGenetics

Achim A WernerORCID

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2 papers

Recent·Most cited

Cell autonomous inflammation in VEXAS is mediated by cGAS-STING

Samuel J. Magaziner, Jason C. Collins, Brecca Miller, Patrick Zheng, Amy K. Wang, Jérôme Hadjadj, Juan Carlos Balandrán, Maria Sirenko, Maya English, James Bertlin, Rebecca L. Murray, Peter H. Whitney, Tania J González-Robles, Deborah Rivera, Yan Wang, , David B. Beck

This study identifies cGAS-STING pathway activation as a key mechanism of inflammation in VEXAS, showing that UBA1 mutations disrupt ERAD and mitochondrial homeostasis, leading to STING-driven inflammation.

bioRxiv · May 29, 2026 · PreprintGenetics

Dephosphorylation of the NPR2 guanylyl cyclase contributes to inhibition of bone growth by fibroblast growth factor

Leia C. Shuhaibar, Jerid W. Robinson, Giulia Vigone, Ninna Shuhaibar, Jeremy R. Egbert, Valentina Baena, Tracy F. Uliasz, Deborah Kaback, Siu‐Pok Yee, Robert Feil, Melanie C. Fisher, Caroline N. Dealy, Lincoln Ross Potter, Laurinda A. Jaffe

The paper shows that dephosphorylation of NPR2 reduces cyclic GMP, counteracting bone elongation induced by FGF signaling.

eLife · Dec 4, 2017Peer-reviewedDevelopmental Biology