Author

Luca Magnani

Institute of Cancer ResearchORCID 0000-0002-7534-0785Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:human· 3 Research Focus:cell type annotation· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:clustering· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Alexander Wilkes· 1 shared Almut SchulzeiD· 1 shared An-Jie Ler· 1 shared Angelika Wörner· 1 shared Anthony J. DevliniD· 1 shared Archana P. ThankamonyiD· 1 shared Birgitta E. MichelsiD· 1 shared Chiara GorriniiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCancer Biology

Alexander Wilkes

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

GLYATL1 is associated with metabolic and epigenetic changes and with endocrine resistance in luminal breast cancer

Janina Müller, Emre Sofyalı, Luisa E. Schwarzmüller, Yael Aylon, Eviatar Weizman, Lisa Schlicker, Katherine Kelly, Simone Borgoni, Simin Oz, Cole Stocker, Sara Burmester, Angelika Wörner, Sabine Karolus, Birgitta E. Michels, Daniela Heiss, , Stefan Wiemann

This study identifies GLYATL1 as a mediator of endocrine resistance in luminal breast cancer by promoting metabolic and epigenetic changes that counteract estrogen deprivation.

Clinical Epigenetics · Apr 29, 2026Peer-reviewedCancer Biology

X-CODE: a dual RNA barcoding system for multi-platform clonal tracking and spatial phenotyping

Roberto Rota, Elizabeth Flittner, Matteo Tartagni, Marwane Bourdim, Emma Westlund, James S. McKenzie, Anthony J. Devlin, Archana P. Thankamony, Nicole Pandell, Florian Gabel, Misha Siddiqui, Dalia Rosano, Alexander Wilkes, Ivan Andrew, Ka Lok Choi, , Marco Bezzi

X-CODE enables multi-platform clonal tracking and spatial phenotyping by combining RNA barcoding with sequencing and imaging technologies.

bioRxiv · Feb 11, 2026 · PreprintCancer Biology

Chemically modified CRISPR-Cas9 enables targeting of individual G-quadruplex and i-motif structures, revealing ligand-dependent transcriptional perturbation.

Sabrina Pia Nuccio, Enrico Cadoni, Roxani Nikoloudaki, Silvia Galli, An-Jie Ler, Claudia Sanchez-Cabanillas, Thomas E. Maher, Ella Fan, Dilek Guneri, Gem Flint, Minghui Zhu, Ling Sum Liu, Christopher R. Fullenkamp, Zoë A. E. Waller, Luca Magnani, , Marco Di Antonio

This paper introduces ATENA, a method using chemically modified CRISPR-Cas9 to target specific G-quadruplexes and i-motifs, showing ligand-dependent transcriptional changes in c-MYC.

Nature Communications · Dec 9, 2025Peer-reviewedCell Biology