Author

Kee-Pyo Kim

Max Planck Institute for Molecular BiomedicineORCID 0000-0002-8666-8444Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Developmental Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Hans Robert SchöleriD· 2 shared Anne Baron‐Van EvercooreniD· 1 shared Beatriz García-DíaziD· 1 shared Bechara SaykaliiD· 1 shared Blandine Manot-SailletiD· 1 shared Carmen J. WilliamsiD· 1 shared Carsten Tobias KuenneiD· 1 shared Catherine Nicole Domingo· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersDevelopmental Biology

Hans Robert SchölerORCID

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2 papers

Recent·Most cited

The homeobox transcription factor DUXBL controls exit from totipotency

María Vega-Sendino, Felipe F. Lüttmann, Teresa Olbrich, Yanpu Chen, Carsten Tobias Kuenne, Paula Stein, Desiree Tillo, Grace I. Carey, Jiasheng Zhong, Virginia Savy, Lenka Radonova, Tianlin Lu, Bechara Saykali, Kee-Pyo Kim, Catherine Nicole Domingo, , Sergio Ruiz

This study identifies DUXBL as a key factor promoting the silencing of the 2C transcriptional program during exit from totipotency.

Nature Genetics · Mar 20, 2024Peer-reviewedDevelopmental Biology

Multiple sclerosis iPSC-derived oligodendroglia conserve their intrinsic properties to functionally interact with axons and glia in vivo

Sabah Mozafari, Laura Julia Starost, Blandine Manot-Saillet, Beatriz García-Díaz, Yu Kang T. Xu, Delphine Roussel, Marion J. F. Levy, Linda Ottoboni, Kee-Pyo Kim, Hans Robert Schöler, Timothy E. Kennedy, Jack Perry Antel, Gianvito Martino, Marı́a Cecilia Angulo, Tanja Kuhlmann, Anne Baron‐Van Evercooren

The paper shows MS-derived oligodendroglia can myelinate axons and form functional connections in a mouse model, suggesting the differentiation block is due to environmental factors, not intrinsic deficits.

Science Advances · Dec 4, 2020Peer-reviewedNeuroscience