Author

C. Marquis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:drug response / drug discovery· 1 Approach:genetic perturbation / CRISPR· 1

Frequent Co-Authors

H. L. Malaby· 2 shared J. Stumpff· 2 shared A. Blanchard· 1 shared A. J. Bass· 1 shared A. Jones· 1 shared A. Nagaraja· 1 shared C. Koch· 1 shared C. L. Fonseca· 1 shared

Activity by Year

2020-2020 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersCell Biology

H. L. Malaby

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Strongyle-resistant sheep express their potential across environments and leave limited scope for parasite plasticity

G. Salle, V. Deiss, C. Marquis, G. Tosser-Klopp, J. Cortet, D. Serreau, C. Koch, D. Marcon, F. Bouvier, P. Jacquiet, N. Holroyd, A. Blanchard, J. Cotton, M.-M. Mialon, C. Moreno-Romieux

The paper breeds sheep resistant to parasites and finds that resistance remains stable across environments but is affected by specific parasite infections.

bioRxiv · Jun 19, 2020 · PreprintEvolutionary Biology

Chromosomally unstable tumor cells specifically require KIF18A for proliferation

C. Marquis, C. L. Fonseca, K. A. Queen, L. Wood, S. E. Vandal, H. L. Malaby, J. E. Clayton, J. Stumpff

This study shows KIF18A is essential for proliferation of chromosomally unstable tumor cells but not near-diploid cells, with effects linked to centrosome fragmentation and mitotic defects.

bioRxiv · Jun 18, 2020Cell Biology

Selective vulnerability of aneuploid human cancer cells to inhibition of the spindle assembly checkpoint

Y. Cohen-Sharir, J. M. McFarland, M. Abdusamad, C. Marquis, H. Tang, M. R. Ippolito, S. V. Bernhard, K. Laue, H. L. Malaby, A. Jones, M. Kazachkova, N. Lyons, A. Nagaraja, A. J. Bass, R. Beroukhim, , U. Ben-David

This study identifies aneuploid cancer cells as more vulnerable to spindle assembly checkpoint (SAC) inhibition and explores the underlying mechanisms.

bioRxiv · Jun 18, 2020Cancer Biology