Author

Simon E. Vidal

Genentech

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 2 Genomics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Assay:ChIP-seq· 2 Assay:DNA methylation· 2 Organism:mouse· 2 Assay:ATAC-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1

Frequent Co-Authors

Matthias StadtfeldiD· 3 shared Effie ApostolouiD· 2 shared Jane A. SkokiD· 2 shared Valentina SnetkovaiD· 2 shared Alexander PolyzosiD· 1 shared Alexis MorrisseyiD· 1 shared Aristotelis TsirigosiD· 1 shared Begüm AydıniD· 1 shared

Activity by Year

2019-2020 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersDevelopmental Biology

Matthias StadtfeldORCID

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3 papers

Recent·Most cited

The BTB transcription factors ZBTB11 and ZFP131 maintain pluripotency by pausing POL II atpro-differentiation genes

Görkem Garipler, Congyi Lu, Alexis Morrissey, Lorena Sofía López-Zepeda, Simon E. Vidal, Begüm Aydın, Matthias Stadtfeld, Uwe Ohler, Shaun Mahony, Neville E. Sanjana, Esteban O. Mazzoni

A CRISPR screen identifies ZBTB11 and ZFP131 as factors maintaining pluripotency by pausing POL II at pro-differentiation genes.

bioRxiv · Nov 23, 2020 · PreprintDevelopmental Biology

EpiMethylTag simultaneously detects ATAC-seq or ChIP-seq signals with DNA methylation

Priscillia Lhoumaud, Gunjan Sethia, Franco Izzo, Theodore Sakellaropoulos, Valentina Snetkova, Simon E. Vidal, Sana Badri, MacIntosh Cornwell, Dafne Campigli Di Giammartino, Kyu‐Tae Kim, Effie Apostolou, Matthias Stadtfeld, Dan Avi Landau, Jane A. Skok

EpiMethylTag combines ATAC-seq or ChIP-seq with bisulfite conversion to simultaneously assess chromatin accessibility/TF binding and DNA methylation.

Genome biology · Nov 21, 2019Peer-reviewedGenomics

Context-dependent requirement of H3K9 methyltransferase activity during cellular reprogramming to iPSCs

Simon E. Vidal, Alexander Polyzos, Jorge Morales-Valencia, Hongsu Wang, Emily Swanzey, Ly-Sha Ee, Bhishma Amlani, Shengjiang Tu, Yixiao Gong, Valentina Snetkova, Jane A. Skok, Aristotelis Tsirigos, Sang Yong Kim, Effie Apostolou, Matthias Stadtfeld

This study shows that reducing EHMT1/2 activity during reprogramming with ascorbic acid impairs iPSC formation and viability, highlighting the role of H3K9 methylation in reprogramming efficiency.

bioRxiv · Jun 18, 2019 · PreprintDevelopmental Biology